4 results match your criteria: "and the Center for Biomedical Neuroscience[Affiliation]"
Brain Behav Immun
November 2015
Department of Pharmacology and the Center for Biomedical Neuroscience in the School of Medicine, University of Texas Health Science Center at San Antonio, San Antonio, TX 78229, United States; Audie L. Murphy VA Hospital, South Texas Veterans Health Care System, San Antonio, TX 78229, United States. Electronic address:
Cognitive dysfunction in depression is a prevalent and debilitating symptom that is poorly treated by the currently available pharmacotherapies. Research over the past decade has provided evidence for proinflammatory involvement in the neurobiology of depressive disorders and symptoms associated with these disorders, including aspects of memory dysfunction. Recent clinical studies implicate inflammation-related changes in kynurenine metabolism as a potential pathogenic factor in the development of a range of depressive symptoms, including deficits in cognition and memory.
View Article and Find Full Text PDFJ Neurophysiol
October 2013
The Department of Physiology and the Center for Biomedical Neuroscience, University of Texas Health Science Center, San Antonio, Texas; and.
Auditory brain stem circuits rely on fast, precise, and reliable neurotransmission to process auditory information. To determine the fundamental role of myelination in auditory brain stem function, we examined the evoked auditory brain stem response (ABR) from the Long Evans shaker (LES) rat, which lacks myelin due to a genetic deletion of myelin basic protein. In control rats, the ABR evoked by a click consisted of five well-defined waves (denoted waves I-V).
View Article and Find Full Text PDFExp Neurol
February 2007
Department of Pharmacology, and the Center for Biomedical Neuroscience, UTHSCSA, USA.
This study examined the effects of dehydration and rehydration with water on Fos and FosB staining in the brainstem of rats. Male rats were water deprived for 48 h (Dehyd, n=7) or 46 h followed by 2 h access to water (Rehyd, n=7). Controls had ad libitum access to water (Con, n=9).
View Article and Find Full Text PDFExp Neurol
July 2005
Department of Pharmacology and the Center for Biomedical Neuroscience, University of Texas Health Sciences Center at San Antonio, 7703 Floyd Curl Drive, San Antonio, TX 78229, USA.
We studied the effects of osmotic stimulation on the expression of FosB and c-Fos in the supraoptic nucleus (SON) and paraventricular nucleus (PVN). Adult male rats were divided into two groups that were injected with lidocaine (0.1-0.
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