4 results match your criteria: "University of California San Francisco. Electronic address: brett.ley@ucsf.edu.[Affiliation]"
Rebuttal From Dr Ley.
Chest
August 2019
Department of Pulmonary and Critical Care Medicine, Kaiser Permanente San Francisco, San Francisco, CA; Department of Medicine, University of California San Francisco, CA. Electronic address:
COUNTERPOINT: Should Molecular and Genetic Biomarkers Be Used in the Initial Evaluation of Patients With Fibrotic ILD? No.
Chest
August 2019
Department of Pulmonary and Critical Care Medicine, Kaiser Permanente San Francisco, and the Department of Medicine, University of California San Francisco. Electronic address:
Prevalence and Clinical Significance of Antineutrophil Cytoplasmic Antibodies in North American Patients With Idiopathic Pulmonary Fibrosis.
Chest
October 2019
Department of Internal Medicine, University of California San Francisco, San Francisco, CA; Department of Pulmonary and Critical Care Medicine, Kaiser Permanente San Francisco, San Francisco, CA. Electronic address:
Background: Antineutrophil cytoplasmic antibodies (ANCAs) have been reported to occur in 7% to 10% of patients with idiopathic pulmonary fibrosis (IPF), but their clinical relevance remains unclear. The aim of this study was to estimate the prevalence of ANCAs in a North American population with IPF and evaluate their clinical significance.
Methods: This was a retrospective study of two independent cohorts of patients diagnosed with IPF at the University of California San Francisco (discovery cohort) and the University of Chicago (replication cohort).
The MUC5B promoter polymorphism and telomere length in patients with chronic hypersensitivity pneumonitis: an observational cohort-control study.
Lancet Respir Med
August 2017
Department of Medicine, University of California San Francisco, San Francisco, CA, USA.
Background: Patients with hypersensitivity pneumonitis are at risk of developing pulmonary fibrosis, which is associated with reduced survival. In families with multiple affected members, individuals might be diagnosed as having idiopathic pulmonary fibrosis (IPF) or chronic (fibrotic) hypersensitivity pneumonitis, which suggests these disorders share risk factors. We aimed to test whether the genomic risk factors associated with the development and progression of IPF are also associated with the development of fibrosis and reduced survival in people with chronic hypersensitivity pneumonitis.
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