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N-methyladenosine (mA) methyltransferase METTL3-mediated LINC00680 accelerates osteoarthritis through mA/SIRT1 manner.
Cell Death Discov
May 2022
Department of Joint Surgery, Center for Orthopaedics Surgery, The Third Affiliated Hospital of Southern Medical University (Academy of Orthopaedics Guangdong Province), Tianhe District, Guangzhou, Guangdong Province, China.
Increasing evidence suggest the biological roles of N-methyladenosine (mA) and long noncoding RNAs (lncRNAs) in the bone disease, especially osteoarthritis (OA). However, the interaction of mA and lncRNA in osteoarthritis is still unclear. Here, we found that a mA-related lncRNA LINC00680 upregulated in the OA tissue and IL-1β-induced isolated primary chondrocytes.
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