3 results match your criteria: "The Second Affiliated Hospital of Medical School of Xi'an Jiao Tong University[Affiliation]"

Inhibition of STAT1 sensitizes radioresistant nasopharyngeal carcinoma cell line CNE-2R to radiotherapy.

Oncotarget

February 2018

Department of Radiation Oncology, The Affiliated Tumor Hospital of Guangxi Medical University, Cancer Institute of Guangxi Zhuang Autonomous Region, Key Laboratory of High Incidence Tumor Prevention and Treatment Guangxi Medical University, Ministry of Education, Nanning, 530021, PR China.

Radioresistance remains a major obstacle for clinicians in the treatment of nasopharyngeal carcinoma (NPC). Others and we have reported that signal transducer and activator of transcription 1 (STAT1) may be as an important gene for resistance to radiation. However, the relationship between STAT1 and radioresistance is still elusive.

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Purpose: To study the radionuclide distribution in mandible of patients with facial asymmetry and help to select proper time and type of treatment.

Methods: The control group included 29 people with no facial asymmetry, while the experimental group consist of 207 patients with facial asymmetry. The radioactive count in 29 normal controls and 207 patients with facial asymmetry were measured and compared using 99mTc-MDP intake percentage in mandibular condyle, ramus and body.

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Background: Currently, up-regulated proteins and apoptosis in hepatitis C is a hot topic in exploring the pathogenic mechanism of Heptitis C Virus(HCV). Some recent studies shows that prohibitin is overexpressed in cells expressing HCV core proteins, and up-regulated prohibitin is also found in human hepatoma cell line HCC-M, lung cancer, prostate cancer, and other cancers. Prohibitin is an important member of the membrane protein superfamily, and it plays a role of molecular chaperones in mitochondrial protein stability.

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