6 results match your criteria: "The Centre of Marine Sciences[Affiliation]"
Microbiota Comparison of Amur ide () Intestine and Waters at Alkaline Water and Freshwater as the Living Environment.
Front Microbiol
May 2022
Key Laboratory of Cold Water Fish Germplasm Resources and Multiplication and Cultivation of Heilongjiang Province, Heilongjiang River Fisheries Research Institute of Chinese Academy of Fishery Sciences, Harbin, China.
The intestinal microbiota of marine animals was influenced by the water and environment in which they live. The Amur ide () adapts to extremely high alkalinity and is an ideal material for aquacultural studies of alkaline adaptation. In this study, we screened intestinal indicator flora and functional redundancy of intestinal colonies in alkaline-water species (AW) and freshwater species (FW) of Amur ide () in these different aquatic environments.
View Article and Find Full Text PDFAuthor Correction: Global vulnerability of marine mammals to global warming.
Sci Rep
March 2020
MARBEC, Univ Montpellier, CNRS, Ifremer, IRD, Montpellier, France.
An amendment to this paper has been published and can be accessed via a link at the top of the paper.
View Article and Find Full Text PDFFull annual monitoring of Subantarctic Emiliania huxleyi populations reveals highly calcified morphotypes in high-CO winter conditions.
Sci Rep
February 2020
CCMAR, The Centre of Marine Sciences, Universidade do Algarve, Campus de Gambelas, 8005-139, Faro, Portugal.
Ocean acidification is expected to have detrimental consequences for the most abundant calcifying phytoplankton species Emiliania huxleyi. However, this assumption is mainly based on laboratory manipulations that are unable to reproduce the complexity of natural ecosystems. Here, E.
View Article and Find Full Text PDFGlobal vulnerability of marine mammals to global warming.
Sci Rep
January 2020
MARBEC, Univ Montpellier, CNRS, Ifremer, IRD, Montpellier, France.
Article Synopsis
- Climate change may soon cause more extinctions in marine mammals than habitat loss or overexploitation, threatening vital ecological roles in ocean ecosystems.
- A study identified the North Pacific Ocean, Greenland Sea, and Barents Sea as the most vulnerable regions for marine mammals under both high and low greenhouse gas emission scenarios.
- Key threatened species like the North Pacific right whale and dugong exhibited unique traits, and their potential extinction could lead to significant losses in functional diversity, impacting marine ecosystems significantly.
Chronic Kidney Disease Circulating Calciprotein Particles and Extracellular Vesicles Promote Vascular Calcification: A Role for GRP (Gla-Rich Protein).
Arterioscler Thromb Vasc Biol
March 2018
From the Centre of Marine Sciences (C.S.B.V., L.S., D.C.S.), GenoGla Diagnostics, Centre of Marine Sciences (C.S.B.V., D.C.S.), and Department of Biomedical Sciences and Medicine (A.P.S., P.L.N.), University of Algarve, Faro, Portugal; UCIBIO-REQUIMTE, Departamento de Química, Faculdade de Ciências e Tecnologia, Universidade NOVA de Lisboa, Caparica, Portugal (A.L.M., R.M.); Centro de Investigação Interdisciplinar Egas Moniz, Egas Moniz-Cooperativa de Ensino Superior CRL, Caparica, Portugal (A.A.M.); Nephrology Department, Centro Hospitalar do Algarve, Faro, Portugal (A.P.S., P.L.N.); VIB-UGent Center for Medical Biotechnology Center and UGent Department of Biochemistry, Ghent, Belgium (A.S., K.G.); and R&D Group VitaK (C.V.) and Department of Biochemistry - Vascular Aspects, Faculty of Medicine, Health and Life Science (L.S.), Maastricht University, The Netherlands.
Objective: Inhibition of mineral crystal formation is a crucial step in ectopic calcification. Serum calciprotein particles (CPPs) have been linked to chronic kidney disease (CKD) calcification propensity, but additional knowledge is required to understand their function, assemblage, and composition. The role of other circulating nanostructures, such as extracellular vesicles (EVs) in vascular calcification is currently unknown.
View Article and Find Full Text PDFGla-rich protein acts as a calcification inhibitor in the human cardiovascular system.
Arterioscler Thromb Vasc Biol
February 2015
From the Centre of Marine Sciences (CCMAR) (C.S.B.V., M.S.R., I.M.L., R.M.C., S.S., S.C., D.C.S.), GenoGla Diagnostics (C.S.B.V., D.C.S.), University of Algarve, Faro, Portugal; Department of Histopathology, Algarve Medical Centre, Faro, Portugal (J.L.E., A.T.); Department of Chemistry, QOPNA, Mass Spectrometry Center, University of Aveiro, Aveiro, Portugal (R.V.); Service of Cardiothoracic Surgery, Santa Cruz Hospital, Centro Hospitalar de Lisboa Ocidental, Lisbon, Portugal (J.N.); UCIBIO@REQUIMTE, Departamento de Química, Faculdade de Ciências e Tecnologia, Universidade Nova de Lisboa, Lisbon, Portugal (A.L.M.); VitaK, Maastricht University, Maastricht, The Netherlands (B.A.G.W., C.V.); and Department of Biochemistry, Cardiovascular Research Institute Maastricht, Maastricht University Medical Centre, Maastricht, The Netherlands (B.A.G.W.).
Objective: Vascular and valvular calcifications are pathological processes regulated by resident cells, and depending on a complex interplay between calcification promoters and inhibitors, resembling skeletal metabolism. Here, we study the role of the vitamin K-dependent Gla-rich protein (GRP) in vascular and valvular calcification processes.
Approach And Results: Immunohistochemistry and quantitative polymerase chain reaction showed that GRP expression and accumulation are upregulated with calcification simultaneously with osteocalcin and matrix Gla protein (MGP).