6 results match your criteria: "Saitama Neurosurgical Institute[Affiliation]"

New Injection Points of Onabotulinum Toxin A for Spastic Paralysis of the Fingers: Eight Cases of Report.

Ann Indian Acad Neurol

June 2020

Department of Neurosurgery, Teikyo University Mizonokuchi Hospital, Takatsu-ku, Kawasaki, Department of Neurosurgery, Saitama Neurosurgical Institute, Konosu, Saitama, Japan.

Objective: Onabotulinum toxin A (botulinum A toxin) is utilized to extend flexed extremities in spastic hemiparesis. Injection points are important to obtain a better effect. Injecting botulinum A toxin into the forearm muscles is a standard method for flexed wrist and fingers; however, we developed new injection points in the intrinsic muscles of the hand to acquire more reliable effect.

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A case of contrecoup skull fracture caused by mild head injury.

Clin Neurol Neurosurg

October 2014

Department of Neurosurgery, Saitama Neurosurgical Institute, 664-1, Kamiya, Kounosu-City, Saitama 365-0027, Japan. Electronic address:

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A 31-year-old female came to our hospital complaining of left frontal bulging with pain on 10 August 2000. The head x-p showed a radiolucent lesion and bulging at the same calvarial site. CT scan and MRI showed fluid-fluid levels, diploic cyst, deformity and hypertrophic calvarial change.

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We encountered three cases of unilateral absence of the external carotid artery. We were able to find only Franklin's case reported in the literature published to date. The absence of the external carotid artery has been explained as due to the fact that the development of the external carotid artery arising from the ventral aorta was disturbed by unknown factors in the early embryonic stage.

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A 66-year-old female presented with spontaneous intraorbital hematoma manifesting as left exophthalmos after left eye discomfort persisting for 5 months. She had no history of head injury. Computed tomography revealed a round mass in her left orbit which was slightly high density with no postcontrast enhancement.

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We studied the lymphokine-activated killer (LAK) activity in splenocytes and thymocytes of rats with brain tumors chronologically from the early stage to the late stage, in order to clarify how much LAK activity would be developed at each stage. Simultaneously the natural killer (NK) activity in splenocytes, as one aspect of the host immunocompetence, was also determined. The splenic NK activity was significantly depressed in rats with brain tumors during the 2nd and 3rd weeks after tumor transplantation, as compared with normal controls.

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