2 results match your criteria: "Poole Hospital and University of Bournemouth[Affiliation]"
Altered C-tactile processing in human dynamic tactile allodynia.
Pain
February 2013
Institute of Neuroscience and Physiology, University of Gothenburg, 413 90 Gothenburg, Sweden Alan Edwards Centre for Research on Pain, McGill University, Montreal, Canada Department of Clinical Neurophysiology, Poole Hospital and University of Bournemouth, Bournemouth, UK Department of Integrative Physiology, School of Medicine, University of Western Sydney, Sydney, Australia.
Human unmyelinated (C) tactile afferents signal the pleasantness of gentle skin stroking on hairy (nonglabrous) skin. After neuronal injury, that same type of touch can elicit unpleasant sensations: tactile allodynia. The prevailing pathophysiological explanation is a spinal cord sensitization, triggered by nerve injury, which enables Aβ afferents to access pain pathways.
View Article and Find Full Text PDFUnmyelinated tactile afferents underpin detection of low-force monofilaments.
Muscle Nerve
July 2006
Department of Clinical Neurophysiology, Poole Hospital and University of Bournemouth, Bournemouth, United Kingdom.
Human hairy but not glabrous skin has unmyelinated (C) tactile (CT) afferents that project to insular cortex. We studied two subjects with the rare sensory neuronopathy syndrome who lack A-beta fibers but have relatively preserved C-fiber function. Weak monofilaments were detected on hairy skin alone.
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