Erratum to '2015 HRS/EHRA/APHRS/SOLAECE expert consensus statement on optimal implantable cardioverter-defibrillator programming and testing' [Journal of Arrhythmia 32/1 (2016) 1-28].

[This corrects the article DOI: 10.1016/j.joa.

[Changes in clinical characteristics and treatment of cerebral venous and sinus thrombosis: analysis of 35 patients admitted in Peking Union Medical College Hospital].

Objective: To investigate the differences in etiology, symptoms, treatment, and prognosis between the patients with cerebral venous and sinus thrombosis (CVST) before December 31 2000 and those after that date.

Methods: The etiology, symptom, treatment, and prognosis of the 35 CVST patients admitted to Peking Union Medical College Hospital (PUMC) from January 1 1991 to December 31 2000 (Group A, n = 16) and those admitted from January 1 2000 to May 31 2007 (Group B, n = 19) were analyzed and compared.

Results: (1) The first two common causes were infection and hematological disease in Group A, while the first two common causes were immune disease and hematological disease in Group B.

[Protection of ulinastatin against myocardial injury induced by off-pump coronary artery bypass graft surgery: report of 24 cases].

Objective: To investigate the protective effect of ulinastatin against myocardial injury induced by off-pump coronary artery bypass graft surgery (OPCAB).

Methods: Twenty-four patients undergoing OPCAB were randomly divided into 2 equal groups: ulinastatin group in which ulinastatin was infused intravenously, firstly at the concentration of 6000 U/kg dissolved in 50 ml normal saline for 30 min and then at the speed of 1000 U.kg(-1).

[Cervical carcinoma in situ: a clinical and pathological analysis of 118 cases].

Objective: To analyze the clinical characteristics and treatment of cervical carcinoma in situ.

Methods: The clinical data of 118 patients with cervical carcinoma in situ (CIS), aged 38.8 (24-77), were retrospectively analyzed.

[The roles of complement 1q and anti-C1q autoantibodies in pathogenesis of lupus nephritis].

Objective: To analyze the correlation of serum levels of complement 1q (C1q) and anti-C1q autoantibodies (C1qAb) with renal pathology in lupus nephritis (LN) and to explore the probable mechanism of C1q and C1qAb in the development of systemic lupus erythematosus (SLE), especially LN.

Methods: The serum samples of 80 SLE patients, 40 LN patients and 40 non-LN patients, were collected to detect the level of C1q with single radial immunodiffusion and the level of C1qAb with enzyme-linked immunosorbent assay. Renal biopsy was conducted in 25 of the 32 LN patients so as to observe the deposition of apoptotic cells and apoptotic bodies in the glomeruli with TdT-mediated dUTP nick end labeling.