4 results match your criteria: "Los Angeles School of Medicine 90095-1662[Affiliation]"
Transcription initiation from TATA-less promoters within eukaryotic protein-coding genes.
Biochim Biophys Acta
March 1997
Department of Microbiology and Immunology, University of California, Los Angeles School of Medicine 90095-1662, USA.
Transcription of the lymphocyte-specific terminal deoxynucleotidyltransferase gene requires a specific core promoter structure.
Proc Natl Acad Sci U S A
April 1996
Howard Hughes Medical Institute, University of California, Los Angeles School of Medicine 90095-1662, USA.
The terminal deoxynucleotidyltransferase (TdT) gene encodes a template-independent DNA polymerase that is expressed exclusively in immature lymphocytes. The TdT promoter lacks a TATA box, but an initiator element (Inr) overlaps the transcription start site. The Inr directs basal transcription and also mediates activated transcription in conjunction with an upstream element called D'.
View Article and Find Full Text PDFCIF, an essential cofactor for TFIID-dependent initiator function.
Genes Dev
April 1996
Howard Hughes Medical Institute, University of California, Los Angeles School of Medicine 90095-1662, USA.
The core promoters for mammalian protein-coding genes often contain a TATA box, an initiator (Inr) element, or both of these control elements. The TFIID complex is essential both for TATA activity and for the activity of a common class of Inr elements characterized by an approximate consensus sequence PyPyA+1NT/APyPy. Although the complete set of proteins required for basal TATA-mediated transcription has been established, the requirements for TFIID-dependent Inr activity remain undefined.
View Article and Find Full Text PDFCombinatorial regulation of transcription II: The immunoglobulin mu heavy chain gene.
Immunity
May 1995
Howard Hughes Medical Institute, Department of Microbiology and Immunology, University of California, Los Angeles School of Medicine 90095-1662, USA.