4 results match your criteria: "Liyang People's Hospital of Jiangsu Province[Affiliation]"

Article Synopsis
  • * A total of 186 videos were evaluated using the Global Quality Score (GQS) and modified DISCERN (mDISCERN) tools, showing a median GQS score of 3 and an mDISCERN score of 2, with health professional videos rated higher than those from individual users.
  • * The findings indicate that only a small percentage of videos adequately covered lung cancer complications, and there is a positive correlation between video quality scores and factors such as video duration.
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Background: Breastfeeding not only meets the nutritional needs of newborn growth and development but also promotes uterine contraction and discharge of lochia, which helps in maternal recovery. However, some mothers experience abnormal lactation and breast swelling due to a lack of breastfeeding knowledge, painful cesarean incisions, anesthesia, negative emotions, and other factors, resulting in a reduced breastfeeding rate, which adversely affects neonatal and maternal health.

Aim: To explore the effects of care intervention with a health education form for breastfeeding on breastfeeding-related conditions.

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The function of long non-coding RNA (lncRNA) nuclear enriched abundant transcript 1 (NEAT1) has been revealed in injury caused by myocardial ischemia/reperfusion (I/R), however, its association with Sevoflurane (Sev), an anesthetic effective for regulating inflammation and oxidative stress, is not yet clear in I/R injury. The aim of this study was to functionally validate and elucidate the mechanism-of-action for Sev-mediated NEAT1 in myocardial I/R injury. Firstly, reduced NEAT1 was revealed in myocardial I/R injured mice treated with Sev.

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MiR-326/Sp1/KLF3: A novel regulatory axis in lung cancer progression.

Cell Prolif

March 2019

Department of Oncology, the First Affiliated Hospital of Nanjing Medical University, Jiangsu Province Hospital, Nanjing, China.

Article Synopsis
  • - The study focuses on Krüppel-like factor 3 (KLF3) and its role in lung cancer, showing that KLF3 levels are higher in lung cancer tissues and that reducing its expression hampers cancer cell growth and induces cell death.
  • - Various assays were used to assess KLF3’s effect on cancer cell behavior, leading to findings that its knockdown limits cell proliferation, migration, and invasion while promoting apoptosis and halting the cell cycle.
  • - The research identifies a regulatory pathway involving miR-326, Sp1, and KLF3, suggesting this axis could be a potential target for new lung cancer treatments.
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