8 results match your criteria: "Keio University Global Research Institute (KGRI)[Affiliation]"
Healthcare (Basel)
November 2024
Department of Health Policy and Management, Graduate School of Medicine, Keio University School of Medicine, Tokyo 160-8582, Japan.
Public Health Nutr
October 2024
Department of Global Health Policy, Graduate School of Medicine, The University of Tokyo, Tokyo, Japan.
BMJ Open
October 2024
Department of Health Policy and Management, School of Medicine, Keio University, Shinjuku-ku, Tokyo, Japan
BMJ Open
September 2024
Department of Global Health Policy, The University of Tokyo, Bunkyo-ku, Tokyo, Japan.
Background: Two decades have passed since the beginning of the Iraq War in 2003. Iraq has long suffered from conflicts and instability, where the people have limited access to healthcare. The coronavirus disease (COVID-19) pandemic brought additional disruption to health service provision.
View Article and Find Full Text PDFJMIR Cancer
August 2024
Department of Surgery, Minamisoma Municipal General Hospital, Minamisoma, Japan.
Neurobiol Stress
September 2022
Keio University Global Research Institute (KGRI), 2-15-45 Mita, Minato-ku, Tokyo, 108-8345, Japan.
Brain and gut microbes communicate in a bidirectional manner with each affecting a person's response to psychosocial stress. Although human studies demonstrated that the intake of probiotics can alter stress-related behavior in both patients and healthy participants, the association between stress-related brain functions and the gut microbiota has mostly been investigated in patients with depression. However, the response to psychosocial stress differs, even among healthy individuals, and elucidating the natural state of the gut microbiota would broaden the understanding of responses to psychosocial stress.
View Article and Find Full Text PDFEndocr J
February 2020
Department of Endocrinology, Metabolism and Nephrology, Keio University School of Medicine, Shinjuku, Tokyo 160-8582, Japan.
Recent studies have revealed that decline in cellular nicotinamide adenine dinucleotide (NAD) levels causes aging-related disorders and therapeutic approaches increasing cellular NAD prevent these disorders in animal models. The administration of nicotinamide mononucleotide (NMN) has been shown to mitigate aging-related dysfunctions. However, the safety of NMN in humans have remained unclear.
View Article and Find Full Text PDFStem Cell Reports
February 2019
Department of Physiology, Keio University School of Medicine, 35 Shinanomachi, Shinjuku-ku, Tokyo 160-8582, Japan. Electronic address:
Non-cell-autonomous effects on neuronal cells are considered to be involved in the pathogenesis of neurodegenerative diseases but have yet to be mechanistically proven. In this issue of Stem Cell Reports, di Domenico et al. provide direct evidence that α-synuclein transferred from astrocytes exerts non-cell-autonomous neuronal dysfunction on dopaminergic neurons in Parkinson's disease (PD).
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