3 results match your criteria: "Jing An Central District Hospital[Affiliation]"
JCAD deficiency delayed liver regenerative repair through the Hippo-YAP signalling pathway.
Clin Transl Med
March 2024
Department of Medical Microbiology & Parasitology, MOE/NHC/CAMS Key Laboratory of Medical Molecular Virology, School of Basic Medical Sciences, Fudan University Shanghai Medical College, Shanghai, China.
Article Synopsis
- After liver surgery, the liver takes longer to heal, and researchers are looking for ways to help it recover faster.
- This study focused on a protein called JCAD and how it affects liver regeneration after surgery in special mice.
- The results showed that without JCAD, liver cells struggled to grow and divide properly, which could help doctors find new treatments for patients after liver transplants.
IgM anti-preS2 monitoring during combined corticosteroid/interferon-alpha 2b therapy in chronic hepatitis B.
Liver
December 1991
Clinical Immunology Research Unit, Jing An Central District Hospital, Shanghai, China.
The biological significance of antibody reactivities towards the preS2 gene encoded proteins of HBV is not yet well known. We investigated the pretreatment IgM anti-preS2 (1-55) ad reactivity in 22 Chinese and 11 Swedish patients with active HBV disease. Significantly enhanced IgM anti-preS2 levels (titers greater than 1/1000) were observed in 48% (16/33) of these patients.
View Article and Find Full Text PDFThe interaction between native serum albumin and hepatitis B virus.
Arch Virol
April 1988
Clinical Immunology Research Unit, Jing An Central District Hospital, Shanghai, China.
Purified hepatitis B virus particles were obtained from HBeAg positive serum by sucrose gradient ultracentrifugation and sephadex G-200 gel filtration. These virions formed a precipitation line in counterimmune electrophoresis with anti-albumin antibody, but the reaction could be inhibited by anti-HBs. After two months at 4 degrees C, another precipitating line was formed under the same condition which could not be inhibited by anti-HBs and was, thus, due to free albumin.
View Article and Find Full Text PDF