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Background: Psoriasis is a noncontagious auto-inflammatory chronic skin disease. So far, some of the inflammatory genes were upregulated in mouse model of psoriasis. This study examined changes in skin mRNA expression of L-kynureninase (), cathelicidin antimicrobial peptide (), beta-defensin 2 (), and proenkephalin () in a mouse model of imiquimod-induced psoriasis.

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