3 results match your criteria: "Health and Insurance Hitoyoshi General Hospital[Affiliation]"
Cerebral thrombosis in a postmenopausal woman on HRT.
J Clin Neurosci
January 2005
Department of Neurosurgery, Health and Insurance Hitoyoshi General Hospital, Japan.
We report stroke in a 62-year-old woman who had been on hormone replacement therapy (HRT), estrogen plus progestin, for more than 8 years. She experienced two episodes of transient ischemic attack (TIA), manifesting as right-sided weakness, that persisted for 10-20 min. Magnetic resonance angiography (MRA) obtained before complete stroke, demonstrated severe stenosis of the left middle cerebral artery, without evidence of cerebral infarction.
View Article and Find Full Text PDFDiffusion weighted image negative transient ischaemic attack and reversible ischaemic neurological deficit. A report of 10 patients with complete recovery.
J Clin Neurosci
August 2004
Department of Neurosurgery, Health and Insurance Hitoyoshi General Hospital, Japan.
Diffusion-weighted magnetic resonance imaging (DWI MRI)-negative transient ischaemic attack (TIA) in hyperacute stroke is a well-known clinical entity. However, no precise analysis of this phenomenon has been reported. We identified 10 patients with TIA or reversible ischaemic neurological deficits (RIND) with no focal hyperintensity on DWI MRI among 108 consecutive acute stroke patients who underwent DWI MRI.
View Article and Find Full Text PDFMR imaging findings of spinal posterior column involvement in a case of Miller Fisher syndrome.
AJNR Am J Neuroradiol
April 2004
Department of Neurosurgery, Health and Insurance Hitoyoshi General Hospital, Kumamoto, Japan.
Article Synopsis
- The case describes a 43-year-old man diagnosed with Miller Fisher syndrome (MFS) who experienced rapid onset symptoms including ptosis, eye muscle paralysis, and severe ataxia.
- Initial MRI scans revealed lesions in the cauda equina, leading to the diagnosis and subsequent immunotherapy treatment, which improved some symptoms but not all.
- A follow-up MRI five months later showed lesions in the spinal posterior column, suggesting they were affecting the posterior nerve roots and contributing to the persistent severe ataxia.