6 results match your criteria: "General Medicine Center and University of Electronic Science and Technology[Affiliation]"
Corrigendum to "HMGB1-TLR4-IL-23-IL-17A axis accelerates renal ischemia-reperfusion injury via the recruitment and migration of neutrophils" [Int. Immunopharmacol. 94 (2021) 107433].
Int Immunopharmacol
March 2022
General Medicine Center and University of Electronic Science and Technology, Sichuan Academy of Sciences & Sichuan Provincial People's Hospital, Chengdu 610072, China. Electronic address:
Corrigendum to "HMGB1-TLR4-IL-23-IL-17A axis accelerates renal ischemia-reperfusion injury via the recruitment and migration of neutrophils" [Int. Immunopharmacol. 94 (2021) 107433].
Int Immunopharmacol
August 2021
General Medicine Center and University of Electronic Science and Technology, Sichuan Academy of Sciences & Sichuan Provincial People's Hospital, Chengdu 610072, China. Electronic address:
HMGB1-TLR4-IL-23-IL-17A axis accelerates renal ischemia-reperfusion injury via the recruitment and migration of neutrophils.
Int Immunopharmacol
May 2021
General Medicine Center and University of Electronic Science and Technology, Sichuan Academy of Sciences & Sichuan Provincial People's Hospital, Chengdu 610072, China. Electronic address:
Renal ischemia-reperfusion injury (IRI) is an important cause of setting off acute kidney injury. Neutrophil-mediated immunomodulation has a pivotal role in the evolving of IRI. The HMGB1-TLR4-IL-23-IL-17A axis gives rise to neutrophil activation.
View Article and Find Full Text PDFPioglitazone alleviates cisplatin nephrotoxicity by suppressing mitochondria-mediated apoptosis via SIRT1/p53 signalling.
J Cell Mol Med
October 2020
Sichuan Academy of Sciences & Sichuan Provincial People's Hospital, General Medicine Center and University of Electronic Science and Technology, Chengdu, China.
Pioglitazone (PIO) attenuates cisplatin nephrotoxicity whereas the underlying mechanism remains unknown. Apoptosis is associated with mitochondrial dysfunction and SIRT1 activation can decrease cell apoptosis in cisplatin nephrotoxicity. Therefore, we explored whether the protective effect of PIO in cisplatin nephrotoxicity is achieved by suppressing mitochondria-mediated apoptosis through SIRT1/p53 signalling regulation.
View Article and Find Full Text PDFManaging the dialysis mode for people infected with COVID-19.
Ren Fail
November 2020
Department of Nephrology, University of Electronic Science and Technology, Sichuan Academy of Sciences, Sichuan Provincial People's Hospital, Sichuan clinical Research Center for Kidney Disease, Chengdu, China.
The anti-inflammatory effects of curcumin on renal ischemia-reperfusion injury in rats.
Ren Fail
November 2018
c General Medicine Center and University of Electronic Science and Technology , Sichuan Academy of Sciences & Sichuan Provincial People's Hospital, Chengdu , China.
The present study aimed to explore the protective mechanism of curcumin-precondition in renal ischemia-reperfusion (I/R) injury. Thirty male SD rats were randomly equally divided into Sham, IRI, and Curcumin groups (n = 10). The model of IRI was induced by clamping the left renal artery for 45 min followed by 24 h reperfusion with the contralateral nephrectomy.
View Article and Find Full Text PDF