2 results match your criteria: "Dept. of Physiology University of Oslo[Affiliation]"
Preoperative unstable angina causes venous adaptation to surgical graft injury.
Basic Res Cardiol
May 2007
Institute of Basic Medical Science, Dept. of Physiology University of Oslo, 1103 Blindern, 0317, Oslo, Norway.
Ischemic preconditioning may provide a systemic organ protection, evident as the phenomenon known as remote preconditioning. Unstable angina may be a clinical analogue to ischemic preconditioning. Vein graft harvesting induces inflammation of the graft wall.
View Article and Find Full Text PDFMyocardial protection evoked by hyperoxic exposure involves signaling through nitric oxide and mitogen activated protein kinases.
Basic Res Cardiol
July 2007
Institute of Basic Medical Science, Dept. of Physiology University of Oslo, Oslo, Norway.
Background: Hyperoxic exposure in vivo (> 95% oxygen) attenuates ischemia-reperfusion injury, but the signaling mechanisms of this cardioprotection are not fully determined. We studied a possible role of nitric oxide (NO) and mitogen activated protein kinases (MAPK) in hyperoxic protection.
Methods: Mice (n = 7-9 in each group) were kept in normoxic or hyperoxic environments for 15 min prior to harvesting the heart and Langendorff perfusion with global ischemia (45 min) and reperfusion (60 min).