Lipid peroxidation and antioxidant enzymes in erythrocytes and tissues in aged diabetic rats.

Antidiabetic treatment with powdered fruit of Capparis decidua decreased alloxan induced lipid peroxidation (LPO) significantly in erythrocytes, kidney and heart. Erythrocyte superoxide dismutase (SOD) activity decreased while the kidney and heart SOD increased in diabetic animals. These alterations in SOD were counteracted by insulin as well as with powdered fruit of C.

Cadmium-induced lipid peroxidation and the status of the antioxidant system in rat tissues.

Cadmium may induce oxidative damage in different tissues by enhancing peroxidation of membrane lipids and altering the antioxidant system of the cells. The peroxidative damage to the cell membrane may cause injury to cellular components due to the interaction of metal ions with the cell organelles. The treatment with Cd (0.

Down regulation of photosynthesis in Artabotrys hexapetatus by high light.

Using variable to maximum fluorescence (Fv/Fm) as the criterion, the down regulation of photosynthesis by high light stress was characterized in the detached leaves of Artabotrys hexapetatus. The decrease in Fv/Fm was corelated with the decrease in oxygen evolution by thylakoids isolated from high light exposed leaves. The decrease in Fv/Fm was linear with increasing time of exposure to high light.

Counteraction by glutathione and selenium of the prooxidant effect of alloxan in erythrocytes in vitro.

Alloxan produces reactive oxygen species which cause injury to the insulin-producing beta-cells of the pancreas. The action of alloxan in the production of free radicals was studied in vitro using rat erythrocytes. Alloxan increased lipid peroxidation (LPO) and decreased the activities of the antioxidant enzymes such as superoxide dismutase, catalase and glutathione peroxidase in the erythrocytes.

Cadmium-induced lipid peroxidation in rat liver slices: A possible involvement of hydroxyl radicals.

Cadmium-induced lipid peroxidation (LPO) in the presence of various hydroxyl radical-generating media was studied in rat liver slices. In the absence of such media, Cd showed an inhibitory action on LPO in vitro. Cd-induced LPO in liver slices in the presence of OH radical-generating media was inhibited by antioxidants.

Effect of glucose on lipid peroxidation and antioxidant enzyme activities in rat liver slices.

Glucose induced lipid peroxidation in rat liver slices and caused membrane damage to liver cells due to oxidative stress. Superoxide dismutase provided an antioxidant enzyme protection against the oxidative stress at low glucose concentrations (25-75 mm) but catalase showed no significant changes. The activity of both of these enzymes decreased in the liver slices at a higher concentration of glucose (150 mm).

Photoprotection of ultraviolet light irradiated E. Coli. B/r cells.

Ultraviolet irradiated E. Coli. B/r cells recover from UV damage when the cells are kept in dark due to dark repair mechanism.

Zinc protection against lipid peroxidation from cadmium.

Effect of zinc and cadmium on lipid peroxidation and catalase activity in liver, heart, brain and testis was determined in order to characterise the interaction of zinc with cadmium. Zinc and cadmium both increased lipid peroxidation significantly in the tissues studied. In animals pretreated with zinc prior to cadmium administration, significant decrease in lipid peroxidation in liver was observed.