2 results match your criteria: "Beijing Anzhen Hospital Affiliated the Capital Medical University[Affiliation]"
Association between RNF41 gene c.-206 T > A genetic polymorphism and risk of congenital heart diseases in the Chinese Mongolian population.
Genet Mol Res
June 2016
The Central Laboratory, Shenzhen Second People's Hospital, Shenzhen University First Affiliated Hospital, Shenzhen, China.
This study aimed to explore the association between ring finger protein 41 (RNF41) c.-206 T > A variant and susceptibility to congenital heart disease (CHD) in the Chinese Mongolian population. The association between RNF41 gene c.
View Article and Find Full Text PDFMuscle-specific deletion of comparative gene identification-58 (CGI-58) causes muscle steatosis but improves insulin sensitivity in male mice.
Endocrinology
May 2015
Departments of Biochemistry (P.X., Y.M., F.G., L.Y.) and Anesthesiology (L.G.), Wake Forest School of Medicine, Winston-Salem, North Carolina 27157; Department of Animal and Avian Sciences (A.K.G.G., Y.M., L.Y.), University of Maryland, College Park, Maryland 20742; Diabetes and Obesity Research Center (X.H., M.W.), Sanford-Burnham Medical Research Institute, Orlando, Florida 32827; Department of Biology (B.X., H.S.), Georgia State University, Atlanta, Georgia 30303; and The Key Laboratory of Remodeling-Related Cardiovascular Diseases (H.L.), Capital Medical University, Ministry of Education, Beijing Institute of Heart, Lung, and Blood Vessel Diseases, Beijing Anzhen Hospital Affiliated the Capital Medical University, Beijing 100029, People's Republic of China.
Intramyocellular accumulation of lipids is often associated with insulin resistance. Deficiency of comparative gene identification-58 (CGI-58) causes cytosolic deposition of triglyceride (TG)-rich lipid droplets in most cell types, including muscle due to defective TG hydrolysis. It was unclear, however, whether CGI-58 deficiency-induced lipid accumulation in muscle influences insulin sensitivity.
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