3 results match your criteria: "48463 Kerman University of Medical Sciences[Affiliation]"
Recent advances on brain drug delivery via nanoparticles: alternative future materials for neuroscience applications; a review.
Rev Neurosci
January 2025
Neuroscience Research Center, Institute of Neuropharmacology, Kerman University of Medical Science, Kerman, Iran.
Essentially, the blood-brain barrier (BBB) serves as a line of demarcation between neural tissues and the bloodstream. A unique and protective characteristic of the blood-brain barrier is its ability to maintain cerebral homeostasis by regulating the flux of molecules and ions. The inability to uphold proper functioning in any of these constituents leads to the disruption of this specialized multicellular arrangement, consequently fostering neuroinflammation and neurodegeneration.
View Article and Find Full Text PDFSocial, academic, and emotional self-efficacies in adolescent girls and their determinants: a cross sectional study.
Int J Adolesc Med Health
December 2024
Nursing Research Center, 48463 Kerman University of Medical Sciences, Kerman, Iran.
Article Synopsis
- The study aimed to assess social, academic, and emotional self-efficacy in school-aged girls and identify contributing factors in Sistan and Baluchistan, Iran.
- It involved 246 girls aged 11 to 18, using various questionnaires to measure self-efficacy, anxiety, and depression.
- Results indicated that all self-efficacy types were above average, with anxiety, education level, and age identified as significant predictors, suggesting that reducing anxiety and depression could improve self-efficacy.
The nongenomic neuroprotective effects of estrogen, E2-BSA, and G1 following traumatic brain injury: PI3K/Akt and histopathological study.
Horm Mol Biol Clin Investig
March 2024
Pathology and Stem Cell Research Center, 48463 Kerman University of Medical Sciences, Kerman, Iran.
Objectives: Studies suggest that both genomic and nongenomic pathways are involved in mediating the salutary effects of steroids following traumatic brain injury (TBI). This study investigated the nongenomic effects of 17β-estradiol (E2) mediated by the PI3K/p-Akt pathway after TBI.
Methods: Ovariectomized rats were apportioned to E2, E2-BSA (E2 conjugated to bovine serum albumin), G1 [G-protein-coupled estrogen receptor agonist (GPER)] or their vehicle was injected following TBI, whereas ICI (classical estrogen receptor antagonist), G15 (GPER antagonist), ICI + G15, and their vehicles were injected before the induction of TBI and injection of drugs.