5 results match your criteria: ""Vasile Goldis" West University Arad[Affiliation]"
Int J Gen Med
September 2024
Department of Cardiology, "Victor Babes" University of Medicine and Pharmacy, Timisoara, 300041, Romania.
Background: Over the years, troponins have aced the para-clinical tests for confirming the diagnosis of acute myocardial infarction. However, the rise in their levels is entirely time-dependent, which can cause a delay in the initiation of treatment protocols. Heart fatty acid binding protein (H-FABP) can serve comparatively as a better biological marker for overcoming this flaw of troponins, as it is quickly released into the bloodstream once the myocardial injury occurs due to decreased blood supply.
View Article and Find Full Text PDFLife (Basel)
August 2024
Discipline of Internal Medicine and Ambulatory Care, Prevention and Cardiovascular Recovery, Department of VI Cardiology, "Victor Babeș" University of Medicine and Pharmacy, 300041 Timișoara, Romania.
J Clin Med
August 2024
Department V, Internal Medicine I-Discipline of Medical Semiology I, Center of Advanced Research in Cardiology and Hemostaseology, "Victor Babeș" University of Medicine and Pharmacy, Eftimie Murgu Sq. no. 2, 300041 Timișoara, Romania.
Background: Galectin-3 has been reported as a mediator of heart failure (HF) development and progression. Most studies, however, have been conducted on patients with chronic HF rather than acute HF (AHF). The aim of this study was to confirm galectin-3 as a prognostic marker in subjects with AHF and to investigate its possible relationship with left ventricular (LV) remodeling.
View Article and Find Full Text PDFBackground: Heart failure is accompanied by abnormalities in ventricular-vascular interaction due to increased myocardial and arterial stiffness. Galectin-3 is a recently discovered biomarker that plays an important role in myocardial and vascular fibrosis and heart failure progression.
Objectives: The aim of this study was to determine whether galectin-3 is correlated with arterial stiffening markers and impaired ventricular-arterial coupling in decompensated heart failure patients.