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Dipeptidyl Peptidase 4 (DPP4) Exacerbates Osteoarthritis Progression in an Enzyme-Independent Manner.
Adv Sci (Weinh)
December 2024
Department of Orthopaedic Surgery, Orthopaedic Research Institute, West China Hospital, West China Medical School, Sichuan University, Chengdu, 610041, China.
Chondrocyte senescence is a key driver of osteoarthritis (OA). Mitochondrial dysfunction and oxidative stress can induce chondrocyte senescence. However, the specific mechanisms by which senescence contributes to OA progression are not fully understood.
View Article and Find Full Text PDFMeasurement of fission product yields in the quasi-mono-energetic neutron-induced fission of U.
Appl Radiat Isot
November 2024
Nuclear Physics Division, Bhabha Atomic Research Centre, Mumbai, 400 085, India.
The cumulative yield of the fission products within the mass range of 83-117 and 123-153 have been measured in the 6.61and 10.92 MeV quasi-mono-energetic neutron-induced fission of U by using an off-line γ-ray spectrometric technique.
View Article and Find Full Text PDFRosiglitazone-induced white adipocyte browning is regulated by actin and Myh9.
Life Sci
December 2024
Key Laboratory of Agriculture Animal Genetics, Breeding and Reproduction of the Ministry of Education, College of Animal Science and Technology, Huazhong Agricultural University, Wuhan, Hubei 430070, China; Frontiers Science Center for Animal Breeding and Sustainable Production, Wuhan, Hubei 430070, China; Hubei Hongshan Laboratory, Wuhan, Hubei 430070, China. Electronic address:
Aims: This study investigates the role of actin polymerization and Myh9 in mediating lipid droplet (LD) fission during rosiglitazone-induced browning of white adipocytes. The aim is to understand how LD splitting might contribute to the beige conversion of white adipose tissue, providing insights into adipocyte plasticity and metabolic regulation.
Materials And Methods: C3H10 T1/2-differentiated adipocytes were used as a classical model to study white adipocyte browning.
Temporal expression of mitochondrial life cycle markers during acute and chronic overload of rat plantaris muscles.
Front Physiol
August 2024
Department of Exercise Science, University of Puget Sound, Tacoma, WA, United States.
Skeletal muscle hypertrophy is generally associated with a fast-to-slow phenotypic adaptation in both human and rodent models. Paradoxically, this phenotypic shift is not paralleled by a concomitant increase in mitochondrial content and aerobic markers that would be expected to accompany a slow muscle phenotype. To understand the temporal response of the mitochondrial life cycle (i.
View Article and Find Full Text PDFEicosapentaenoic Acid (EPA) and Docosahexaenoic Acid (DHA) Ameliorate Heart Failure through Reductions in Oxidative Stress: A Systematic Review and Meta-Analysis.
Antioxidants (Basel)
August 2024
Faculty of Medicine, University of British Columbia, 9th Floor 2775 Laurel St., Vancouver, BC V5Z 1M9, Canada.
The objectives of this study were to explore the role that eicosapentaenoic acid (EPA) and/or docosahexaenoic acid (DHA) plays in heart failure (HF), highlighting the potential connection to oxidative stress pathways. Following PRISMA guidelines, we conducted electronic searches of the literature in MEDLINE and EMBASE focusing on serum EPA and/or DHA and EPA and/or DHA supplementation in adult patients with heart failure or who had heart failure as an outcome of this study. We screened 254 studies, encompassing RCTs, observational studies, and cohort studies that examined HF outcomes in relation to either serum concentrations or dietary supplementation of EPA and/or DHA.
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