The purpose of the present study was to investigate whether or not low-intensity ultrasound exposure (20 mW/cm2 average intensity) accelerates the repair of rat germinal epithelium damaged by an antispermatogenic agent. The results from analysis of testicular weight and DNA content, sperm production and epididymal sperm concentration showed that the time needed for the reestablishment of the spermatogenic process following busulfan treatment was not reduced by ultrasound energy suggesting that, in contrast to many other mammalian tissues, the seminiferous epithelium is refractory to ultrasound stimulation.
Download full-text PDF |
Source |
---|---|
http://dx.doi.org/10.1016/s0301-5629(97)00185-3 | DOI Listing |
Enter search terms and have AI summaries delivered each week - change queries or unsubscribe any time!