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[Tuberculous cranial pachymeningitis presenting with long-standing diffuse brain dysfunction].
Rinsho Shinkeigaku
April 2011
Department of Neurology, Showa University School of Medicine.
We report a 59-year-old immunocompetent man presenting with slowly progressive gait unsteadiness, dysarthria, and clumsiness in writing over 6 months. There were bilateral pyramidal signs, pseudobulbar palsy, and attention deficits. Cerebrospinal fluid examination showed mild mononuclear pleocytosis, and magnetic resonance imaging revealed pachymeningeal pattern of contrast enhancement beneath the calvarium and the posterior cranial fossa.
View Article and Find Full Text PDFThe results of clinical, neuropsychological and MRI study of 21 patients with "ephedron" encephalopathy caused by intake of methcatinon ("ephedron"), a surrogate drug obtained from phenylpropanolamine-containing compounds by adding potassium permanganate, are presented. Signs of brain lesions emerged 3-14 (mean 6.8 +/- 4.
View Article and Find Full Text PDF[Gustatory disturbance associated with putaminal hemorrhage].
Rinsho Shinkeigaku
August 2002
5th Department (Neurology) of Internal Medicine, Fukuoka University School of Medicine.
48-year-old left-handed man experienced a sudden loss of taste, followed by dysarthria and dysphagia. Taste threshold examined by a small filter-paper disc method was severely elevated on both sides of the tongue. Brain CT revealed right putaminal hemorrhage that measured 9.
View Article and Find Full Text PDF[Paucisymptomatic and reversible myelinolysis after an anaphylactic shock].
Rev Neurol (Paris)
November 2002
Service de Neurologie, CHU Henri MONDOR, Créteil, France.
Article Synopsis
- - Myelinolysis is a type of demyelination primarily affecting the central part of the pons, leading to symptoms like spastic tetraparesis and pseudobulbar paralysis.
- - The case study involves a 58-year-old man who experienced pontine and extrapontine myelinolysis following an anaphylactic shock, despite not having low sodium levels (hyponatremia).
- - The patient's condition improved over a few days, and MRI results normalized within three months, suggesting that mild myelinolysis can resolve on its own after a shock event.
[The role of immunologic reactions in the pathogenesis in Binswanger's disease; a clue to therapeutic approach].
Rinsho Shinkeigaku
January 1999
Department of Neurology, Faculty of Medicine, Kyoto University.
We examined the alterations of glial cells in the brains with Binswanger's disease. In comparison to the brains with lacunar cerebral infarction and those from non-neurological controls, oligodendroglia was decreased in number, and microglia was increased and activated in the white matter lesions in Binswanger's disease. Astroglia occasionally showed a regressive change, termed clasmatodendrosis.
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