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Anti-Phospholipase A2 Receptor in Nonlupus Patients with Membranous Nephropathy and Crescents.
Glomerular Dis
April 2022
Department of Pathology, Microbiology, and Immunology, Vanderbilt University Medical Center, Nashville, Tennessee, USA.
Introduction: Anti-phospholipase A2 receptor (PLA2R) is detected in approximately 70% of biopsies of "primary" membranous nephropathy (MN). Crescents in MN in nonlupus patients suggest additional injury, such as antineutrophil cytoplasmic antibody (ANCA) or anti-glomerular basement membrane (anti-GBM)-associated glomerulonephritis and are postulated to reflect injury by a mechanism that unmasks cryptic epitopes leading to the second autoantibody.
Methods: We studied PLA2R staining in nonlupus patients with MN and crescents.
Pulmonary renal syndrome: A case report of diffuse alveolar hemorrhage in association with ANCA negative pauci-immune glomerulonephritis.
Medicine (Baltimore)
June 2018
Division of Pulmonary and Critical Care Medicine Department of Medicine, Bronx Care Health System, Bronx, NY Department of Pathology, College of Physicians and Surgeons, Columbia University Affiliated with Icahn School of Medicine at Mount Sinai, New York, NY.
Rationale: Pulmonary renal syndrome (PRS) is a term most commonly used to describe a combination of glomerulonephritis and pulmonary hemorrhage as a manifestation of a multisystem autoimmune disease. It is usually associated with ANCA vasculitis and anti-GBM disease. Diffuse alveolar hemorrhage in a patient with ANCA and anti-GBM negative pauci-immune glomerulonephritis is rare and optimal management is unknown.
View Article and Find Full Text PDFRenal-limited vasculitis with elevated levels of multiple antibodies.
CEN Case Rep
May 2017
Department of Nephrology, Graduate School of Medicine, Kyoto University, 54 Shogoin Kawahara-cho, Sakyo, Kyoto, 606-8507, Japan.
Renal-limited vasculitis (RLV) is a type of anti-neutrophil cytoplasmic antibody (ANCA)-associated vasculitis that presents with crescentic glomerulonephritis with no other organ involvement. Although several studies reported patients with crescentic glomerulonephritis who were dual positive for proteinase 3 (PR3)-ANCA and myeloperoxidase (MPO)-ANCA or ANCA and anti-glomerular basement membrane (GBM) antibody, patients positive for all three antibodies, i.e.
View Article and Find Full Text PDF[An experience of treatment of double positive myeloperoxidase-antineutrophil cytoplasmic antibodies (MPO-ANCA) and anti-glomerular basement membrane antibodies in Goodpasture's syndrome onset of crescentic glomerulonephritis].
Nihon Jinzo Gakkai Shi
November 1998
Third Department of Internal Medicine, Kinki University School of Medicine, Osaka, Japan.
A 68-year-old woman was admitted to Kinki University Hospital because of progressive renal failure. She had been well until two months before admission. Laboratory data were as follows: serum creatinine 4.
View Article and Find Full Text PDFAnti-GBM-associated crescentic glomerulonephritis with discrete IgG deposition, but with no electron-dense material in glomeruli.
Nephrol Dial Transplant
October 1996
First Department of Medicine, Teikyo University, School of Medicine, Tokyo, Japan.
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