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Erythrokinetic mechanism(s) causing the "late anemia" of hemolytic disease of the fetus and newborn.
J Perinatol
June 2024
Division of Hematology/Oncology, Department of Pediatrics, University of Utah, Salt Lake City, UT, USA.
A transfusion-requiring "late anemia" can complicate the management of neonates convalescing from hemolytic disease of the fetus and newborn (HDFN). This anemia can occur in any neonate after HDFN but is particularly prominent in those who received intrauterine transfusions and/or double-volume exchange transfusions. Various reports describe this condition as occurring based on ongoing hemolysis, either due to passive transfer of alloantibody through breast milk or persistence of antibody not removed by exchange transfusion.
View Article and Find Full Text PDFBackground: Systemic lupus erythematosus and anti-phospholipid syndrome may cause severe haematological complications despite few other symptoms of disease.
Case Presentation: A previously healthy woman in her late twenties was admitted to hospital with chest pain and dyspnoea. CT of the thorax revealed bilateral pulmonary embolism and urine sampling showed haematuria and proteinuria.
A computational study of structural differences of binding of NADP and G6P substrates to G6PD Mediterranean, G6PD A-, G6PD Cairo and G6PD Gaza mutations.
Blood Cells Mol Dis
July 2021
Division of Hematology, Department of Internal Medicine, University of Utah School of Medicine, Salt Lake City, UT, United States of America. Electronic address:
Glucose-6-phosphate dehydrogenase (G6PD) deficiency is a common X-linked inherited enzymopathic disorder that may lead to transfusion-requiring acute hemolytic anemia (AHA) triggered by fava beans ingestion, infection or some drugs. The gene encoding for G6PD carries a large number of genetic variants that have varying pathogenicity. We reported on three G6PD variants in the Gaza Strip Palestinian population with differing clinical impacts and frequencies: G6PD Mediterranean, African G6PD A-, and G6PD Cairo.
View Article and Find Full Text PDFAnti-Lan Antibodies: A Rare Etiology of Severe Blood Transfusion Reaction.
Cureus
October 2020
Medical Oncology, East Tennessee State University, Johnson City, USA.
Lan is a high prevalence red blood cell antigen present in the majority of the populations that belong to the Lan (Langereis) blood group system. Anti-Lan antibody is an immunoglobulin G (IgG) antibody that is known to cause delayed hemolytic transfusion reactions in adults as well as hemolytic disease in fetuses and newborns, however with variable clinical significance ranging from mild to severe. We present a 58-year-old woman with diffuse abdominal pain and a large gastric ulcer causing gastric outlet obstruction.
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