AI Article Synopsis

  • - A 74-year-old male with non-insulin-dependent diabetes mellitus (NIDDM) was hospitalized due to vision issues and was found to have dangerously low calcium and parathyroid hormone levels.
  • - Brain scans showed abnormal calcifications in areas like the basal ganglia and cerebellum.
  • - After treating his low calcium levels, the patient's insulin production returned to normal, suggesting that hypocalcemia may have contributed to his diabetes by impacting insulin secretion, alongside a genetic predisposition.

Article Abstract

We report a case of non-insulin-dependent diabetes mellitus (NIDDM) complicated with idiopathic hypoparathyroidism. A 74-year-old male was hospitalized because of diplopia. He was revealed to have NIDDM. The levels of serum Ca and intact-PTH were 6.3 mg/dl and < 5 pg/ml, respectively. Brain computed tomography revealed abnormal calcification in the cerebral basal ganglia and the cerebellum. After recovery from hypocalcemia, the endogenous insulin secretion was normalized. It is suggested that the pathogenesis of NIDDM in this patient may have been related to an insulin secretory defect as a result of hypocalcemia in addition to the hereditary risk.

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http://dx.doi.org/10.2169/internalmedicine.34.904DOI Listing

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