The zone of the bronchial constriction of the esophagus and the area of the upper and lower dilatation of the esophagus was studied in humans of different age by the morphohistotopographic method at the macro- and microscopic levels. The bronchial constriction area of the esophagus was found to have a thickening of the circular layer of the muscular tunic, less relative thickness of the longitudinal layer, changed course of muscular fibers. The smooth muscle thickening of the circulating layer of the muscular tunic is localized at the boundary of the transitional zone of the displacement of the striated muscle fibers by smooth myocytes. The above morphological signs are characteristic of the areas with sphincter functions which allows to consider the bronchial constriction as a sphincter zone of the esophagus.
Download full-text PDF |
Source |
|---|
Publication Analysis
Top Keywords
Similar Publications
Lobar lung transplantation, followed by partial sternal resection and bronchial stenosis, in a patient with scoliosis.
J Cardiothorac Surg
January 2025
Prague Lung Transplant Program, 3rd Department of Surgery, First Faculty of Medicine, Charles University and Motol University Hospital, Prague, Czech Republic.
Scoliotic deformity represents a serious spinal disorder that influences the locomotive and cardiopulmonary systems. Some patients with severe scoliosis and end-stage lung disease are therefore denied lung transplantation. In patients with scoliosis considering lung transplantation, size match, straight back syndrome, delayed chest closure and bronchial stenosis are key issues clinicians should evaluate.
View Article and Find Full Text PDFBackground: In patients with asthma, bronchoconstriction and airway inflammation both contribute to airway narrowing and airflow limitations, which lead to symptoms and exacerbations. Short-acting beta 2-agonist (SABA)-only rescue therapy addresses only bronchoconstriction and is associated with increased morbidity and mortality. Current asthma management guidelines recommend concomitant treatment of symptoms and inflammation with a fast-acting bronchodilator and inhaled corticosteroid (ICS) as rescue therapy for patients 12 years of age.
View Article and Find Full Text PDFEfficacy and safety of long-acting muscarinic antagonists in COPD: a meta-analysis and meta-regression with a focus on aging.
Respir Med
January 2025
Respiratory Disease and Lung Function Unit, Department of Medicine and Surgery, University of Parma, Parma, Italy.
The increasing global elderly population, projected to reach 20% of individuals aged 65 and over by 2030, faces significant pulmonary challenges, including chronic obstructive pulmonary disease (COPD). Aging is associated with a natural decline in lung function and structural changes that exacerbate respiratory issues. COPD, characterized by chronic respiratory symptoms and airflow obstruction, presents a unique challenge in older patients due to the accelerated decline in lung function.
View Article and Find Full Text PDFLoss of Iron Regulatory Protein 2 (IRP2) Protects Mice from Lung Inflammation during Bronchoconstriction.
Am J Respir Cell Mol Biol
January 2025
Max Planck Institute of Biochemistry, Immunoregulation, Martinsried, Germany;
Formaldehyde and asthma: a plausibility?
Arch Toxicol
January 2025
National Research Centre for the Working Environment, Copenhagen, Denmark.
Formaldehyde (FA) is a ubiquitous indoor air pollutant emitted from construction, consumer, and combustion-related products, and ozone-initiated reactions with reactive organic volatiles. The derivation of an indoor air quality guideline for FA by World Health Organization in 2010 did not find convincing evidence for bronchoconstriction-related reactions as detrimental lung function. Causal relationship between FA and asthma has since been advocated in meta-analyses of selected observational studies.
View Article and Find Full Text PDFWant AI Summaries of new PubMed Abstracts delivered to your In-box?
Enter search terms and have AI summaries delivered each week - change queries or unsubscribe any time!