Introduction: We describe our experience in the treatment of supraventricular tachycardia due to atrioventricular nodal reentry by radiofrequency catheter ablation of the slow pathway.
Patients And Methods: Thirty-four patients (mean age 52 +/- 13 years) with recurrent drug refractory atrioventricular nodal reentrant tachycardia underwent radiofrequency catheter ablation of slow-pathway as the first procedure. Maximal energy used was 28 +/- 8 W, mean time was 4.4 +/- 2.8 min, and a mean number of 13 +/- 9 discharges per patient.
Results: Of the 34 patients 14 were successfully treated (no slow-pathway conduction, no AV nodal reentrant echo complexes, no inducible tachycardia), 11 were considered as partially successful (no inducible tachycardia, with slow-pathway conduction or echoes) and 9 patients were unsuccessfully treated (all of them underwent fast-pathway ablation with a successful outcome). Eight recurrences were observed during a mean follow-up period of 7 +/- 4 months: One in a patient with previously successful ablation (remained asymptomatic while taking beta-blockers), 6 in patients with first procedure partially successful (all of them underwent effective second slow-pathway ablation) and 1 in a patient with an unsuccessful procedure that underwent fast pathway ablation. A repeated procedure was successful in ablating the slow pathway. None of our patients developed complete atrio-ventricular block. One patient developed cardiac tamponade that needed surgical drainage. At last follow-up all patients are free from tachycardias.
Conclusion: Slow pathway ablation is an effective method of treatment in patients with atrio-ventricular nodal reentrant tachycardia. Long term success is related to complete abolition of slow pathway conduction.
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We report a case of long RP' tachycardia diagnosed as fast-slow atrioventricular nodal reentrant tachycardia (AVNRT) with a bystander nodoventricular pathway (NVP). Differential responses to right ventricular extrastimuli from the base and apex highlighted the anatomical proximity of the NVP attachment, contributing to the diagnosis.
View Article and Find Full Text PDFJ Arrhythm
February 2025
Department of Electrophysiology, Department of Cardiology AIG Institute of Cardiac Sciences and Research Hyderabad India.
Objectives: We present a case series of patients with granulomatous myocarditis presenting as atrial arrhythmias accompanied by lymphadenopathy.
Background: Atrial myocarditis (AM) may be the cause of atrial fibrillation (AF) in patients without risk factors.
Methods: Patients with atrial fibrillation without risk factors underwent 18F-Fluorodeoxyglucose positron emission tomography (18F-FDG-PET).
J Cardiovasc Electrophysiol
January 2025
Division of Cardiology, Section of Cardiac Electrophysiology, University of California San Francisco, San Francisco, California, USA.
We encountered a single case in which a transition between orthodromic reciprocating tachycardia with a concealed nodoventricular pathway and atrioventricular nodal reentrant tachycardia with a bystander nodoventricular pathway was observed.
View Article and Find Full Text PDFHeart Rhythm O2
December 2024
Department of Cardiovascular Medicine, Kyorin University School of Medicine, Mitaka, Japan.
Background: Junctional rhythm (JR) frequently occurs during radiofrequency (RF) ablation procedures targeting the slow pathway (SP) for atrioventricular nodal re-entrant tachycardia (AVNRT), signaling successful ablation. Two types of JR have been noticed: typical JR as His activation preceding atrial activation, and atypical JR as atrial activation preceding the His activation. Nevertheless, the origin and characteristics of JR remain incompletely defined.
View Article and Find Full Text PDFJ Cardiovasc Electrophysiol
January 2025
First Department of Cardiology, TEDA International Cardiovascular Hospital, Tianjin, China.
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