Atrioventricular nodal reentrant tachycardia (AVNRT) is the most frequent supraventricular tachycardia. The reentry circuit had been said to be localized within the AV node for many years and the first non pharmacological therapy was the surgical or catheter ablation of the AV node. This was, however, too high a price for a generally well tolerated and non life-threatening arrhythmia. Only recently, the endocardial mapping and the results of surgical perinodal dissection showed that part of the reentry circuit was localized in the atrial myocardium near the AV node. The first approach was the ablation of the fast pathway as it is easier to map during AVNRT. However, this pathway is located very close to the AV node, so that its ablation is complicated by a percentage of AV block that is too high (6.2%) considering the good prognosis of this arrhythmia. In order to reduce this risk, the ablation of the slow pathway which is located more posteriorly and more distant from the AV node, was then proposed. Three different approaches have been suggested; one purely anatomic and the other two guided by electrophysiologic markers. If the posterior and middle part of the septum during sinus rhythm is mapped, more posteriorly, near the coronary sinus os, the sharp potential, described by Jackman, is recorded. It is a sharp spike with a high amplitude, associated with an atrial electrogram of very low amplitude. It cannot be modified by atrial pacing and may also be recorded during the uncommon form of AVNRT.(ABSTRACT TRUNCATED AT 250 WORDS)
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We report a case of long RP' tachycardia diagnosed as fast-slow atrioventricular nodal reentrant tachycardia (AVNRT) with a bystander nodoventricular pathway (NVP). Differential responses to right ventricular extrastimuli from the base and apex highlighted the anatomical proximity of the NVP attachment, contributing to the diagnosis.
View Article and Find Full Text PDFJ Arrhythm
February 2025
Department of Electrophysiology, Department of Cardiology AIG Institute of Cardiac Sciences and Research Hyderabad India.
Objectives: We present a case series of patients with granulomatous myocarditis presenting as atrial arrhythmias accompanied by lymphadenopathy.
Background: Atrial myocarditis (AM) may be the cause of atrial fibrillation (AF) in patients without risk factors.
Methods: Patients with atrial fibrillation without risk factors underwent 18F-Fluorodeoxyglucose positron emission tomography (18F-FDG-PET).
J Cardiovasc Electrophysiol
January 2025
Division of Cardiology, Section of Cardiac Electrophysiology, University of California San Francisco, San Francisco, California, USA.
We encountered a single case in which a transition between orthodromic reciprocating tachycardia with a concealed nodoventricular pathway and atrioventricular nodal reentrant tachycardia with a bystander nodoventricular pathway was observed.
View Article and Find Full Text PDFHeart Rhythm O2
December 2024
Department of Cardiovascular Medicine, Kyorin University School of Medicine, Mitaka, Japan.
Background: Junctional rhythm (JR) frequently occurs during radiofrequency (RF) ablation procedures targeting the slow pathway (SP) for atrioventricular nodal re-entrant tachycardia (AVNRT), signaling successful ablation. Two types of JR have been noticed: typical JR as His activation preceding atrial activation, and atypical JR as atrial activation preceding the His activation. Nevertheless, the origin and characteristics of JR remain incompletely defined.
View Article and Find Full Text PDFJ Cardiovasc Electrophysiol
January 2025
First Department of Cardiology, TEDA International Cardiovascular Hospital, Tianjin, China.
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