AI Article Synopsis

  • Previous research indicates that coronary collaterals may help prevent stress-induced heart perfusion issues, but their impact on stress echocardiography had not been explored until this study.
  • The study involved 21 patients who underwent both stress echocardiography and coronary angiography, revealing that vessels with angiographic collaterals showed fewer stress-induced wall motion abnormalities compared to those without collaterals.
  • The results suggest that the presence of collateral blood flow can protect the heart muscle, even when there is significant blockage in the coronary arteries.

Article Abstract

Previous studies using thallium-201 scintigraphy have suggested that angiographic coronary collaterals can protect against the development of stress-induced perfusion abnormalities, but the effect of collaterals on stress echocardiography (SECHO) has not been determined. In this study, 21 consecutive patients referred for cardiac catheterization underwent SECHO and coronary angiography. Of the 21 study patients, there was a total of 16 significantly obstructed coronary arteries (> or = 70% stenosis) in 14 patients. SECHO revealed stress-induced wall motion abnormalities in the distribution of seven of nine obstructed coronary vessels without angiographic collaterals, but in only one of seven vessels with collaterals (p < 0.05). Six of eight obstructed vessels not associated with a stress-induced wall motion abnormality had collaterals, whereas only one of eight obstructed vessels associated with a stress-induced wall motion abnormality had collaterals. We conclude that (1) angiographically demonstrated coronary collaterals can protect against the development of stress-induced wall motion abnormalities despite the presence of a high-grade coronary artery obstruction, and (2) the lack of a stress-induced wall motion abnormality on SECHO in the perfusion territory of an obstructed vessel may suggest the presence of adequate collateral perfusion.

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http://dx.doi.org/10.1002/clc.4960180405DOI Listing

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