Download full-text PDF |
Source |
|---|
Publication Analysis
Top Keywords
Similar Publications
Neurotoxic phospholipase A: A proposed cause of equine grass sickness and other animal dysautonomias (multiple system neuropathies).
Equine Vet J
January 2025
National Research Council Institute of Neuroscience and Department of Biomedical Sciences, University of Padova, Padova, Italy.
A possible cause and therapy for equine grass sickness.
Toxicon
July 2024
Microbiology and Veterinary Diagnostic Laboratory, Istituto Zooprofilattico Sperimentale delle Venezie, 35020, Legnaro, Italy. Electronic address:
Update on the equine grass sickness biobank, database and research project.
Vet Rec
June 2024
Moredun Research Institute, Pentlands Science Park, Penicuik, Midlothian, EH26 OPZ.
Beth Wells, equine grass sickness project lead at the Moredun Research Institute, encourages vets to report any equine grass sickness cases they encounter and explains how case reports and samples are being used to support research into this complex disease.
View Article and Find Full Text PDFSupplemental trace minerals as complexed or inorganic sources for beef cattle during the receiving period.
J Anim Sci
January 2024
Department of Animal Science, University of Arkansas System Division of Agriculture, Fayetteville, AR 72701, USA.
To investigate effects of inorganic or complexed trace mineral source (zinc, copper, manganese, and cobalt) on receiving period performance and morbidity, crossbred beef heifer calves (n = 287) arriving on three delivery dates were used in a 42-d receiving trial. Heifers were processed after arrival, stratified by day -1 body weights (BW) and allocated randomly to eight pens (11 to 13 heifers/pen, 24 pens total). Within truckload, pens were assigned randomly to dietary treatment (n = 12 pens/treatment).
View Article and Find Full Text PDFEquine grass sickness is associated with major abnormalities in the ultrastructure of skeletal neuromuscular junctions.
Equine Vet J
January 2025
Medical Toxicology Centre and Institute of Neuroscience, Newcastle University, Faculty of Medical Sciences, Newcastle upon Tyne, UK.
Background: Equine grass sickness (EGS) is a frequently fatal multisystem neuropathy of equids. The aetiology is unknown; proposed causes include toxicoinfection with Clostridium botulinum and a mycotoxicosis. The effect of EGS on the organisation and structural integrity of the skeletal neuromuscular junction (NMJ), the target of botulinum neurotoxins (BoNTs), is unknown.
View Article and Find Full Text PDFWant AI Summaries of new PubMed Abstracts delivered to your In-box?
Enter search terms and have AI summaries delivered each week - change queries or unsubscribe any time!