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SARS-CoV-2 S-protein expression drives syncytia formation in endothelial cells.
Sci Rep
January 2025
Department of Biomedical Engineering, The Ohio State University, Columbus, OH, USA.
SARS-CoV-2 is a viral infection, best studied in the context of epithelial cell infection. Epithelial cells, when infected with SARS-CoV-2 express the viral S-protein, which causes host cells to fuse together into large multi-nucleated cells known as syncytia. Because SARS-CoV-2 infections also frequently present with cardiovascular phenotypes, we sought to understand if S-protein expression would also result in syncytia formation in endothelial cells.
View Article and Find Full Text PDFReply to: On the giant deformation and ferroelectricity of guanidinium nitrate.
Nat Commun
January 2025
Smart Materials Lab, New York University Abu Dhabi, PO Box 129188, Abu Dhabi, UAE.
On the giant deformation and ferroelectricity of guanidinium nitrate.
Nat Commun
January 2025
Faculty of Chemistry, Adam Mickiewicz University, Poznań, Poland.
Mechanics of cell sheets: plectin as an integrator of cytoskeletal networks.
Open Biol
January 2025
Laboratory of Integrative Biology, Institute of Molecular Genetics of the Czech Academy of Sciences, Prague, Czechia.
Epithelia are multicellular sheets that form barriers defining the internal and external environments. The constant stresses acting at this interface require that epithelial sheets are mechanically robust and provide a selective barrier to the hostile exterior. These properties are mediated by cellular junctions which are physically linked with heavily crosslinked cytoskeletal networks.
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