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[Syndrome of anti-basal membrane antibody].
Rev Assoc Med Bras (1992)
February 2005
Disciplina de Nefrologia da Universidade Federal de São Paulo-UNIFESP-EPM, SP.
[The nephrotoxic effects of single duck immunoglobulins in the triggering of Masugi nephritis].
Zentralbl Pathol
July 1992
St. Josef-Hospital, Universitätsklinik Bochum, Deutschland.
Studies were conducted into the roles played by various duck immunoglobulins in the anti-basal membrane of nephrotoxic nephritis in rabbit. The 7.8S and 5.
View Article and Find Full Text PDF[Anti-basal membrane glomerulonephritis after homologous kidney transplantation in hereditary Alport's nephropathy].
Dtsch Med Wochenschr
November 1991
Abteilung für Nephrologie, Universität Düsseldorf.
A patient, born in 1968, was found at the age of 9 years to have Alport's syndrome. In his 20th year, when in terminal renal failure, his father's kidney was transplanted into him. 14 months later there was a sudden worsening of his renal function after a varicella infection; for the first time antibodies against the glomerular basal membrane (GBM) were detected.
View Article and Find Full Text PDFAntisera to basal lamina and glial endfeet disturb the normal extension of axons on retina and pigment epithelium basal laminae.
Development
October 1989
Friedrich Miescher-Institut, Basel, Switzerland.
In order to determine the role of the extracellular matrix in regulating the directed growth of embryonic neurites, antisera to retina (a-RBL I and II), to pigment epithelium (a-PBL) and to glomerular (a-GBL) basal lamina were probed for an effect on the ordered extension of neurites. In the assays, retina explants from chick and quail were cultured on basal lamina from embryonic chick retina and pigment epithelium either in the presence of anti-basal lamina antisera or in the presence of the corresponding preimmune sera. In the presence of all anti-basal lamina antisera, normal extension of axons was greatly inhibited both on retina and on pigment epithelium basal lamina.
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