Three successful cases of reconstruction of the inferior vena cava (one of the bifurcation) by polytetrafluoroethylene grafts suggest further trial of this solution for a difficult and hitherto unsolved problem.
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Biomarkers.
Alzheimers Dement
December 2024
Department of Medical Physics, University of Wisconsin-Madison, Madison, WI, USA.
Background: The spread of tau in Alzheimer's Disease (AD) can be tracked in vivo using [F-18]MK6240, a PET radioligand that binds to tau aggregates in AD with high affinity. However, significant MK6240 signal is also observed in the meninges and sinus and the extra cerebral binding (ECB) signal from these regions can spill into exterior brain regions complicating evaluation of early stage AD tauopathy. This study evaluates the magnitude and variability of ECB in a large imaging cohort to identify trends in this signal.
View Article and Find Full Text PDFBackground: Alzheimer's disease (AD) is multifactorial, thus multivariate analyses help untangle its effects. We employed multiple contrast MRI to reveal age-related brain changes in populations at risk for AD, due to APOE4 carriage. We assessed volume and microstructure changes using diffusion weighted imaging, and quantitative magnetic susceptibility maps (QSM) reflective primarily of cerebral iron metabolism.
View Article and Find Full Text PDFBiomarkers.
Alzheimers Dement
December 2024
Université Paris-Saclay, CEA, CNRS, Inserm, BioMaps, Orsay, France.
Background: Typical Alzheimer's disease (AD) and Limbic-predominant Age-related TDP-43 Encephalopathy (LATE) are two neurodegenerative diseases that present with a similar initial amnestic clinical phenotype but have distinct proteinopathies. AD is characterised by ß-amyloid plaques and intraneuronal neurofibrillary tangles, while LATE is characterised by abnormal neuronal TDP-43 protein. With reference to the prion-like hypothesis regarding the propagation of proteinopathies, investigating white matter fibre bundle alterations could provide new insights into the propagation pathways of specific proteinopathies.
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Alzheimer's Imaging Consortium.
Alzheimers Dement
December 2024
Department of Medical Physics, University of Wisconsin-Madison, Madison, WI, USA.
Background: The spread of tau in Alzheimer's Disease (AD) can be tracked in vivo using [F-18]MK6240, a PET radioligand that binds to tau aggregates in AD with high affinity. However, significant MK6240 signal is also observed in the meninges and sinus and the extra cerebral binding (ECB) signal from these regions can spill into exterior brain regions complicating evaluation of early stage AD tauopathy. This study evaluates the magnitude and variability of ECB in a large imaging cohort to identify trends in this signal.
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