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http://dx.doi.org/10.1016/s0750-7658(83)80052-5 | DOI Listing |
World Neurosurg
January 2025
Department of Neurosurgery, Hospital Nacional Dos de Mayo, Lima, Peru.
Introduction: Microsurgery for paraclinoid aneurysms remains the first line of treatment in resource-constrained settings. The authors describe their institutional experience and evaluate functional outcomes after microsurgical treatment of paraclinoid aneurysms.
Methods: A retrospective review of clinical records was conducted.
Asian J Neurosurg
December 2024
Department of Neurosurgery, Pandit Bhagwat Dayal Sharma Post Graduate Institute of Medical Sciences, Rohtak, Haryana, India.
Cureus
August 2024
Department of Neurosurgery, Taipei Veterans General Hospital, Taipei, TWN.
While osteoporosis is the primary cause of vertebral compression fractures (VCFs), it's crucial to promptly recognize pathological fractures through comprehensive diagnostic tests, including vertebral biopsies, to determine the exact etiology. For instance, a 66-year-old male with osteoporosis experienced worsening lower limb weakness and back pain after an initial vertebroplasty for a T12 compression fracture. Subsequent MRI revealed severe circumferential extradural compression at T12, leading to further surgeries that eventually uncovered metastatic adenocarcinoma from a pancreatic tumor.
View Article and Find Full Text PDFAsian J Neurosurg
September 2024
Department of Neurosurgery, Faculty of Medicine, Menoufia University, Menoufia, Egypt.
This study was a retrospective study conducted from October 2020 to October 2022 on 106 posttraumatic patients with acute extradural hematomas (EDHs) who were initially planned for conservative treatment. 74 patients had spontaneous EDH regression (EDHR), while 32 patients developed EDH progression (EDHP) and were shifted for surgery. The two groups were statistically compared regarding the different demographic, clinical, and radiographic factors to identify the significant predictors for regression versus progression of acute posttraumatic EDH.
View Article and Find Full Text PDFMol Syndromol
August 2024
Division of Human Genetics, Cincinnati Children's Hospital Medical Center, Cincinnati, OH, USA.
Introduction: Lateral meningocele syndrome (LMS), also known as Lehman syndrome, is caused by pathogenic variants in exon 33 of . Variants in this final exon of interrupt the regulatory PEST domain, leading to enhanced signaling due to prolonged cellular half-life. Individuals with LMS are expected to have multiple lateral meningoceles, developmental delay, neonatal hypotonia, dysmorphic facial features, and feeding difficulties.
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