In seven healthy volunteers insulin-induced hypoglycemia caused a significant rise in free serum cationic trypsin-like immunoreactivity (CTLI), which could be blocked by atropine. Atropine alone significantly decreased the serum CTLI level. The inhibitory effect of atropine was augmented by insulin, which appears to be a peripheral inhibitor of the CTLI release from the pancreatic acinar cells.
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