Perfusion of the basal membrane of the Lorenzini ampullae with 10(-4)-10(-6) M L-glutamic amino-acid, irrespective of the concentration of the amino-acid, produced reversible changes in resting firing rate depending only on initial resting rate. D-glutamic and L-aspartic acids exerted inhibitory effects of the electrical activity. The findings are consistent with the opinion that L-glutamic acid is the synaptic transmitter in the Lorenzini ampullae.
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