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Demonstrating the protective effect of a 70-year-old occupational exposure limit against pneumoconiosis caused by mica.
Toxicol Ind Health
February 2022
114866Integral Consulting Inc., Portland, ME, USA.
Workers involved in crushing, milling, screening, and bagging of mica scrap are at increased risk to develop pneumoconiosis, a progressive material overloading of the lung that can lead to fibrosis and, in the later stages, to dyspnea. Pneumoconiosis is only seen after 10-20 years of respiratory mica exposure, and it can have a latency period of up to 40 years-today's cases date back to exposures during the second half of the 20 century. An occupational lifetime exposure level of 3 mg/m respirable mica dust has been considered to present no risk of pneumoconiosis since 1951 when the American Conference of Governmental Industrial Hygienists (ACGIH) established a 20 million particles per cubic foot (mppcf) (3.
View Article and Find Full Text PDF[Pulmonary siderosis: report of a case].
J Formos Med Assoc
December 1993
Department of Internal Medicine, Taipei Municipal Chung-Hsiao Hospital, Taiwan, R.O.C.
Pulmonary siderosis is one kind of pneumoconiosis caused by the long term inhalation of iron dust. It occurs in a number of occupations including steel rolling and grinding, welding, polishing, casting, boiler scaling, iron ore mining and emery working. Here we report a case of pulmonary siderosis.
View Article and Find Full Text PDFA patient with a history of decorating glass fixtures by means of abrasive etching presented with a disease characterized by diffuse infiltrates on chest roentgenogram and restriction and diffusion impairment on pulmonary function testing. Mineralogic analysis of biopsied pulmonary lesions showed particles consistent in composition to the glasses etched and abrasives used. The former particles were noncrystalline.
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