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Motor modules are largely unaffected by pathological walking biomechanics: a simulation study.
J Neuroeng Rehabil
January 2025
Department of Mechanical and Aerospace Engineering, University of Florida, PO Box 116250, Gainesville, FL, 32611, USA.
Background: Motor module (a.k.a.
View Article and Find Full Text PDFUpregulated FoxO1 promotes arrhythmogenesis in mice with heart failure and preserved ejection fraction.
Nat Commun
January 2025
Smidt Heart Institute, Cedars-Sinai Medical Center, Los Angeles, California, CA, USA.
Myocardial fibrosis leads to cardiac dysfunction and arrhythmias in heart failure with preserved ejection fraction (HFpEF), but the underlying mechanisms remain poorly understood. Here, RNA sequencing identifies Forkhead Box1 (FoxO1) signaling as abnormal in male HFpEF hearts. Genetic suppression of FoxO1 alters the intercellular communication between cardiomyocytes and fibroblasts, alleviates abnormal diastolic relaxation, and reduces arrhythmias.
View Article and Find Full Text PDFFabrication of Chitosan/PEO/Rosmarinic acid based nanofibrous mat for diabetic burn wound healing and its anti-bacterial efficacy in mice.
Int J Biol Macromol
January 2025
Department of Zoology, University of Kalyani, Kalyani, Nadia, West Bengal 741235, India. Electronic address:
The pathophysiological relationship between wound healing impairment and diabetes is an intricate process. Burn injury among diabetes patients leads to neurological, vascular, and immunological abnormalities along with impaired activities of cell proliferation, collagen production, growth factors, and cytokine activities with huge bacterial infestation. In our study, we aimed to achieve a burn wound dressing material with the help of electrospun Chitosan/Polyethylene oxide/Rosmarinic acid (CS/PEO/RA) nanofibers.
View Article and Find Full Text PDFUltrasonographic Changes in the Anterior Scalene Muscle in Neurogenic Thoracic Outlet Syndrome.
J Vasc Surg
January 2025
Baylor University Medical Center, Department of Anesthesia, Division of Anesthesia.
Introduction: Thoracic outlet syndrome (TOS) encompasses multiple symptoms produced by compression of the neurovascular bundle within the thoracic outlet. The subtypes of thoracic outlet are termed for the major affected structure including neurogenic (nTOS), venous (vTOS), and arterial (aTOS) thoracic outlet syndrome. Neurogenic thoracic outlet syndrome accounts for over 95 percent of thoracic outlet syndrome cases and occurs from compression of the brachial plexus.
View Article and Find Full Text PDFImproving myocardial infarction diagnosis with Siamese network-based ECG analysis.
PLoS One
January 2025
School of Computer Science & Engineering (SCOPE), VIT-AP University, Amaravati, Andhra Pradesh, India.
Background: Heart muscle damage from myocardial infarction (MI) is brought on by insufficient blood flow. The leading cause of death for middle-aged and older people worldwide is myocardial infarction (MI), which is difficult to diagnose because it has no symptoms. Clinicians must evaluate electrocardiography (ECG) signals to diagnose MI, which is difficult and prone to observer bias.
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