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Objectives: Serum-ascites albumin gradient (SAAG) has been used extensively in the diagnostic workup of patients with ascites. A SAAG level of <1.1 g/dl is usually thought of as a result of nonportal hypertension etiologies, including malignancies, tuberculous peritonitis, and nephrotic syndrome.

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As many as 195 patients were studied presenting with mixed, nephrotic, and hematuric varieties of chronic glomerulonephritis (ChG), aged 6-15 years, including 118 children from the test-groups who were given a combined therapy involving differentiated rest-and-movement regimens (DRMR) and graded therapeutical physical training (GTPT) and 77 controls who received none of the above treatments. The examination involved clinical as well as functional tests (the unit "Cardiocid"-aided ECG, EchoCG on the "Combison-420-5"), with the renal function being evaluated as recommended by Ota Shiuk. Commonly seen in those being admitted into hospital was nephrogenous hypertension as were abnormal changes in myocardium associated with automatism malfunction, inadequate conductivity, and impaired myocardial contractile ability.

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Alpha-tocopherol acetate produced a positive effect on the renal function in patients with nephrogenous hypertension due to replenishment of endogenous antioxidants and its regulatory effect on lipid peroxidation. As a result there occurs a reduction of highly-toxic products of lipid peroxidation, improvement of renal microcirculation, reduction of ischemic disorders in the organs of the body.

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The effect of an intravenous calcium gluconate load (10 mg/kg over 5 min) on plasma ionized calcium concentration, parathyroid hormone (PTH), and the rate of urinary excretion of calcium, sodium, and nephrogenous cyclic adenosine monophosphate (NcAMP) was examined in 26 patients with essential hypertension and 27 age- and sex-matched normotensive subjects. Prior to calcium administration hypertensives had lower plasma ionized calcium concentration (P < .01) and higher PTH levels (P < .

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Altogether 16 patients with arterial hypertension (AH) were examined. Of these, 11 were with stenosis of renal arteries, 4 had essential hypertension (EH) and I with nephrogenous parenchymatous AH. To estimate the influence of captopril on the total and separate renal function in patients with and without stenosis of renal arteries and the possibility of the use of the given pharmacological test in the diagnosis of renovascular hypertension (RVH), all the patients underwent renal scintigraphy with 99Tc-DTPA on admission to the hospital and 1-7 days after a single intake of 25 mg captopril per os.

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