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Diagnostic Accuracy of Shear Wave Elastography in Predicting Malignant Origins of Pleural Effusions in Emergency Departments.
Diagnostics (Basel)
January 2025
Odense Respiratory Research Unit (ODIN), Department of Clinical Research, Faculty of Health Sciences, University of Southern Denmark, 5000 Odense, Denmark.
: Ultrasound is a valuable diagnostic tool in the diagnostic work-up of dyspnea and can identify even small pleural effusions. The incorporation of shear wave elastography (SWE) represents a possible tool in stratifying pleural effusions by the risk of underlying malignancy. No previous studies on ultrasound with the incorporation of SWE have been conducted in an emergency department (ED), where such stratification might have a clinical impact by hastening referrals for the diagnostic work-up of underlying malignancy.
View Article and Find Full Text PDFA 7-point evidence-based care discharge protocol for patients hospitalized for exacerbation of COPD: consensus strategy and expert recommendation.
NPJ Prim Care Respir Med
December 2024
Department of Respiratory Medicine, Lilavati Hospital and Research Centre, Mumbai, India.
Acute exacerbations of COPD (ECOPD) are an important event in the life of a COPD patient as it causes significant deterioration of physical, mental, and social health, hastens disease progression, increases the risk of dying and causes a huge economic loss. Preventing ECOPD is therefore one of the most important goals in the management of COPD. Before the patient is discharged after hospitalization for ECOPD, it is crucial to offer an evidence-based care bundle protocol that will help minimize the future risk of readmissions and death.
View Article and Find Full Text PDFBio-Composite Nanogels Based on Chitosan and Hyaluronic Acid for the Treatment of Lung Infections.
Gels
November 2024
Institute of Polymers, Composites and Biomaterials, National Research Council (IPCB-CNR), Viale J.F. Kennedy 54, 80125 Naples, Italy.
Pathogen infections constitute a serious problem in the field of lung diseases, especially in severe conditions such as chronic obstructive pulmonary disease (COPD) and acute respiratory distress syndrome (ARDS). Exacerbations of COPD and ARDS can be significantly influenced by bacterial infections from Pseudomonas aeruginosa and Staphylococcus aureus, which can hasten the decline of lung function. Moreover, the abuse of high-dose antibiotics used to treat obstinate infections is contributing to the growing issue of multidrug resistance (MDR) by microorganisms.
View Article and Find Full Text PDFAssessing the National Health, Education, and Air Quality Benefits of the United States Environmental Protection Agency's School Bus Rebate Program: A Randomized Controlled Trial Design.
Res Rep Health Eff Inst
October 2024
Department of Biostatistics, University of Washington School of Public Health.
Introduction: Approximately 25 million children ride buses to school in the United States. While school buses remain the safest school transport from a traffic accident perspective, older buses can expose students to high levels of diesel exhaust. These exposures can adversely affect health, which might cause missed school days and reduced learning.
View Article and Find Full Text PDFGenetic absence of PD-L1 does not restore CD8 T cell function during respiratory virus infection and delays virus clearance.
J Virol
October 2024
Department of Pediatrics, University of Pittsburgh School of Medicine, UPMC Children's Hospital of Pittsburgh, Pittsburgh, Pennsylvania, USA.
Article Synopsis
- The study investigates how the inhibitory receptor PD-1 and its ligand PD-L1 affect CD8 T cell function during respiratory virus infections like human metapneumovirus and influenza.
- Researchers found that mice lacking PD-L1 had similar CD8 T cell impairments as normal mice, suggesting that PD-L1's role in T cell function may be complex.
- The results indicated that while PD-L2 does not compensate for the absence of PD-L1, PD-L1 deficiency leads to increased expression of other inhibitory receptors, hinting at additional factors contributing to T cell impairment.
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