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Gastric secretion.
Curr Opin Gastroenterol
November 2002
Department of Medicine, Division of Gastroenterology, Medical College of Virginia and McGuire VAMC Richmond, Virginia 23249, USA.
Overlapping neural, hormonal, and paracrine pathways finely regulate gastric acid secretion. In rats and guinea pigs, most of the intrinsic neural innervation to the gastric mucosa originates in the myenteric plexus. In contrast, human stomachs have a clearly defined submucosal plexus that contains a variety of transmitters including nitric oxide, vasoactive intestinal peptide (VIP), gastrin-releasing peptide (GRP), substance P, and calcitonin gene-related peptide (CGRP).
View Article and Find Full Text PDFEffect of peripherally and centrally administered calcitonin on gallbladder emptying in dogs.
J Gastroenterol
June 1997
Department of Physiology, Institute of Occupational Medicine and Environmental Health, Sosnowiec, Poland.
The effect of calcitonin on meal-stimulated gallbladder emptying (GBE) was examined after intravenous (i.v.) and intracerebroventricular (i.
View Article and Find Full Text PDFNeuroendocrine regulation of canine gastric secretion, emptying and blood flow.
J Neuroendocrinol
June 1989
Neurogastroenterology Laboratory, Departments of Medicine, University of Hamburg, Hamburg, FRG and University of California San Diego, San Diego, California, USA.
Abstract Several peptides have been implicated as central nervous system transmitters regulating various peripheral organ systems. This study examined the central nervous system effects of rat corticotrophin-releasing factor (CRF), salmon calcitonin (CT), beta-endorphin (beta-End), neurotensin (NT), rat calcitonin gene-related peptide (CGRP) and bombesin (BOM) on gastric acid secretion, gastric emptying and left gastric artery flow in conscious dogs. All of these peptides, injected into the third cerebral ventricle, significantly inhibited gastric acid secretion but not plasma gastrin concentrations stimulated by a liquid protein meal.
View Article and Find Full Text PDFMeal-stimulated gastrin release and calcitonin secretion.
J Endocrinol Invest
June 1989
Istituto di Clinica Medica Generale e Terapia Medica, University of Parma, Italy.
In order to study the effects of a physiologic meal on calcitonin (CT) secretion we studied 6 normal male volunteers (aged 28-34 yr). Each subject was given, on two separate days, either a mixed meal or 200 ml of distilled water, in random order. Gastrin (G) was effectively stimulated by the meal (F = 8.
View Article and Find Full Text PDFThis study was designed to assess the central nervous system actions of human calcitonin (hCalc) and human calcitonin gene-related peptide (hCGRP) on meal-stimulated gastric acid secretion in awake beagle dogs. hCalc (0.1-1.
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