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Postnatal Serum Total Thyroxine Level Associated with Short- and Long-Term Anthropometric Outcomes in Very Preterm Infants.
Nutrients
May 2022
Department of Pediatrics, National Cheng Kung University Hospital, College of Medicine, National Cheng-Kung University, Tainan 704302, Taiwan.
Thyroxine (T4) importantly regulates the growth of newborns. Compared to fetuses with equivalent gestational ages, very preterm infants (VPIs) often experience relatively low thyroxinemia, with a normal thyroid-stimulating hormone (TSH) concentration < 10 μIU/mL. However, there is continued debate regarding postnatal thyroxine supplementation for VPIs with normal TSH and transitionally low thyroxinemia.
View Article and Find Full Text PDF[Induction and reversibility of thyroid proliferative changes in suckling rats given thiocyanate].
Ann Endocrinol (Paris)
October 2004
Laboratoire de Physiologie Animale, Département des Sciences de la Vie, Faculté des Sciences de Sfax, Route de la Soukra Km 3,5. BP 802,. 3018 Sfax, Tunisie.
Potassium thiocyanate given in the drinking water of pregnant rats led to decreased body weight in their 14-day-old offspring (27%) without altering thyroid weight. Reduction of the suckling rat's body weight could be explained be defective thyroxinemia (38). Plasma FT3 and TSH were unchanged after thiocyanate treatment.
View Article and Find Full Text PDF[TSH determination in central hypothyroidism].
Ann Endocrinol (Paris)
November 1999
Service d'Endocrinologie, Hôpital Bologhine, Bains-Romains, Alger.
Central hypothyroidism is one of the limitations of the use of sensitive TSH assays as first line screen in evaluating thyroid function. Studies on this subject are however scarce. The aim of the present study is to evaluate the usefulness of TSH assay before and after TRH on a large series of patients with central hypothyroidism.
View Article and Find Full Text PDFEarly effects of iodine deficiency on radial glial cells of the hippocampus of the rat fetus. A model of neurological cretinism.
J Clin Invest
June 1997
Unidad de Neuroanatomía del Instituto Cajal, Consejo Superior de Investigaciones Científicas and Facultad de Medicina de la Universidad Autónoma de Madrid, 28002 Madrid, Spain.
The most severe brain damage associated with thyroid dysfunction during development is observed in neurological cretins from areas with marked iodine deficiency. The damage is irreversible by birth and related to maternal hypothyroxinemia before mid gestation. However, direct evidence of this etiopathogenic mechanism is lacking.
View Article and Find Full Text PDFMaternal diabetes mellitus, a rat model for nonthyroidal illness: correction of hypothyroxinemia with thyroxine treatment does not improve fetal thyroid hormone status.
Thyroid
February 1997
Unidad de Endocrinología Molecular, Universidad Autónoma de Madrid, Spain.
Maintenance of normal maternal thyroxinemia prevents severe triiodothyronine (T3) deficiency of the fetus with primary thyroid failure (1). We have studied whether thyroxine (T4) would also protect the fetal brain when maternal hypothyroxinemia is caused by nonthyroidal illnesses. We have used the streptozotocin-induced diabetes mellitus pregnant rat as a model of maternal nonthyroidal illness.
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