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An Interesting Case of Vitamin D-Mediated Severe Hypercalcemia in a Patient With Renal Mass.
JCEM Case Rep
January 2025
Department of Medical Oncology, Dr. Jeyasekharan Medical Trust Hospital & Nursing Home, Nagercoil, Tamilnadu 629003, India.
Hypercalcemia of malignancy (HCM) is the most common cause of hypercalcemia in hospitalized patients. The pathogenesis of HCM is often multifactorial. One of the rare causes of HCM is extra-renal production of 1,25-dihydroxyvitamin D (or calcitriol), which is often seen in patients with lymphoproliferative malignancies.
View Article and Find Full Text PDFChapter 4: Differential diagnosis of primary hyperparathyroidism.
Ann Endocrinol (Paris)
January 2025
Service d'endocrinologie, diabétologie, métabolisme, nutrition, hôpital Huriez, CHU de Lille, 1, rue Polonovski, 59037 Lille cedex, France; Inserm U1190, institut génomique européen pour le diabète, université de Lille, 59000 Lille, France. Electronic address:
The differential diagnosis of primary hyperparathyroidism can be considered clinically, biologically and radiologically. Clinically, primary hyperparathyroidism should be suspected in case of diffuse pain, renal lithiasis, osteoporosis, repeated fracture, cognitive or psychiatric disorder, or disturbance of consciousness. Nevertheless, the differential diagnosis of primary hyperparathyroidism is mainly biological, particularly in atypical forms, which must be differentiated from hypercalcemia with hypocalciuria or non-elevated PTH on the one hand, and from normo-calcemia with elevated PTH, hypophosphatemia or hypercalciuria on the other.
View Article and Find Full Text PDFDe Novo Hypercalcaemia in a Patient With Chronic Hypoparathyroidism.
Cureus
December 2024
Endocrinology, Mallow General Hospital/University College Cork, Cork, IRL.
Calcium Homeostasis in the human body is regulated by hormones, including parathyroid hormone and vitamin D3. Dysfunction in the form of hypoparathyroidism causes hypocalcaemia. In patients treated for primary hypoparathyroidism with activated vitamin D replacement, iatrogenic hypercalcaemia can occur.
View Article and Find Full Text PDFCalcium and vitamin D substitution for hypoparathyroidism after thyroidectomy - how is it continued after discharge from hospital?
Langenbecks Arch Surg
December 2024
Section of Endocrine Surgery, Department of General, Visceral and Transplantation Surgery, University Medical Center, Johannes Gutenberg University Mainz, Langenbeckstraße 1, D-55131, Mainz, Germany.
Article Synopsis
- The study focuses on managing postoperative hypoparathyroidism (HypoPT) after thyroid surgery, which often requires calcium and active vitamin D supplements.
- During a retrospective analysis from March 2015 to December 2023 at UMC Mainz, it was found that only 28.3% of patients continued receiving these supplements post-discharge, despite varying levels of parathyroid hormone (PTH) and calcium.
- The results highlighted inconsistencies in how external practitioners adjust these supplements, suggesting that thyroid surgeons should recommend a structured reduction plan to prevent excessive calcium intake and potential kidney issues.
Characterization of a cohort of patients with hypercalcemia in a tertiary pediatric hospital.
Arch Argent Pediatr
December 2024
Department of Endocrinology, Hospital de Pediatría S.A.M.I.C. Prof. Dr. Juan P. Garrahan, City of Buenos Aires, Argentina.
Introduction. Hypercalcemia is infrequent in pediatrics, of diverse etiology, and with multiorgan morbidity. Objective.
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