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Milk-alkali syndrome due to Caved-S.
J R Soc Med
August 1992
Department of Medicine, Southampton and St Mary's Hospital, Portsmouth.
Campylobacter pylori and non-ulcer dyspepsia. 2. A prospective study in a Swedish population.
Scand J Gastroenterol Suppl
March 1990
Dept. of Clinical Pathology and Cytology, Falun Hospital, Sweden.
In a consecutive prospective series of 186 Swedish persons with the diagnosis of non-ulcer dyspepsia 71.5% were found to have gastritis and/or bulbar duodenitis in endoscopic biopsies. Gastroduodenitis was associated with campylobacter pylori (CP) in 83.
View Article and Find Full Text PDFCampylobacter pylori and gastroduodenal ulcer disease. A prospective study in a Swedish population.
Scand J Gastroenterol Suppl
March 1990
Dept. of Clinical Pathology and Cytology, Falun Hospital, Sweden.
In a consecutive prospective series of 208 Swedish primary peptic ulcer patients, 146 gastric, 55 duodenal and 7 in both sites, gastroduodenitis was found in 97.6% of the cases. The mucosal inflammation was associated with CP in 87% and 91% of the gastric and duodenal ulcer cases respectively.
View Article and Find Full Text PDFDoes Caved-S decrease the gastric ulcer relapse rate during maintenance treatment with ranitidine?
Aliment Pharmacol Ther
December 1987
Endoscopy Unit, Airedale General Hospital, Steeton, Keighley, UK.
Ninety-five patients with healed gastric ulcers were treated with ranitidine 150 mg at night plus either Caved-S two tablets twice daily, or an identical placebo preparation. Treatment was continued for 2 years: the recurrence rate after 1 year was 12.5% for single therapy and 19% for combined therapy, and 30 and 40% respectively after 2 years (differences not significant).
View Article and Find Full Text PDFIn a double blind endoscopically controlled study, 100 patients with gastric ulcers were treated with either ranitidine or ranitidine plus Caved-S. On single therapy, 58% of ulcers were healed at four weeks, 92% at eight weeks and 96% at 12 weeks. Combination therapy did not alter the healing rate, nor improve on the control of dyspeptic symptoms.
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