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A very rare cause of blue finger: A case-based review.
J Scleroderma Relat Disord
October 2023
The Azrieli Faculty of Medicine, Bar-Ilan University, Safed, Israel.
Introduction: Cryofibrinogen is an abnormal, cold-insoluble protein composed of a combination of fibrinogen, fibrin, and fibronectin. Cryofibrinogenemia can be essential (e.g.
View Article and Find Full Text PDFMonoclonal Gammopathy of Thrombotic Significance.
Cancers (Basel)
January 2023
Department of Clinical Therapeutics, School of Medicine, National and Kapodistrian University of Athens, 15772 Athens, Greece.
The current review provides an overview of the thrombotic risk observed in patients with MG who do not otherwise require treatment. We discuss clinical and biomarker studies that highlight the heterogenous hemostatic profile observed in these patients and how knowledge has evolved over the past 20 years. Biomarker studies suggest shared biologic features between multiple myeloma and monoclonal gammopathy of undetermined significance (MGUS), which involves both hypercoagulability and platelet activation.
View Article and Find Full Text PDFIn vitro diagnostics for the medical dermatologist. Part II: Hypercoagulability tests.
J Am Acad Dermatol
August 2021
Division of Dermatology, Department of Internal Medicine, The Ohio State University, Columbus, Ohio. Electronic address:
The skin often provides initial clues of hypercoagulability with features such as livedo reticularis, livedo racemosa, retiform purpura, necrosis, and ulcerations. Because these cutaneous manifestations are nonspecific, laboratory testing is often needed to evaluate for underlying causes of hypercoagulability. Importantly, these disorders are reported to be the most common mimicker, resulting in an erroneous diagnosis of pyoderma gangrenosum.
View Article and Find Full Text PDFClinical and Histopathologic Characteristics of the Main Causes of Vascular Occlusion - Part I: Thrombi.
Actas Dermosifiliogr (Engl Ed)
January 2021
Servicio de Anatomía Patológica, Hospital Universitario de Cruces, Barakaldo, Vizcaya, España.
Vascular occlusion has multiple, diverse clinical manifestations, some of which can have grave consequences for patients. The causes of vascular occlusion are also highly variable, ranging from thrombi triggered by the uncontrolled activation of coagulation mechanisms, on the one hand, to endothelial dysfunction or occlusion by material extrinsic to the coagulation system on the other. In a 2-part review, we look at the main causes of vascular occlusion and the key clinical and histopathologic findings.
View Article and Find Full Text PDFClinical conditions responsible for hyperviscosity and skin ulcers complications.
Clin Hemorheol Microcirc
January 2018
Dipartimento Biomedico di Medicina Interna eSpecialistica, Università di Palermo, Italy.
In this brief review, we have examined some clinical conditions that result to be associated to an altered hemorheological profile and at times accompanied by skin ulcers. This skin condition may be observed in patients with the following condtions, such as primary polycythemic hyperviscosity (polycythemia, thrombocytemia) treated with hydroxyurea, primary plasma hyperviscosity (multiple myeloma, cryoglobulinemia, cryofibrinogenemia, dysfibrinogenemia, and connective tissue diseases), primary sclerocythemic hyperviscosity (hereditary spherocytosis, thalassemia, and sickle cell disease). In addition, it may be present in patients with secondary hyperviscosity conditions such as diabetes mellitus, arterial hypertension, critical limb ischemia and chronic venous insufficiency.
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