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CT-guided Left Stellate Ganglion Cryoneurolysis for Refractory Ventricular Arrhythmias.
Radiology
December 2024
From the Department of Radiology, Division of Interventional Radiology (N.L., N.J.R.), Department of Medicine, Division of Interventional Cardiology (Y.R.), and Department of Medicine, Division of Cardiovascular Medicine (Y.R., G.S., M.G.), UMass Memorial Medical Center and Chan Medical School, 55 Lake Ave N, S2-817A, Worcester, MA 01655; Department of Radiology and Imaging Sciences, Division of Interventional Radiology and Image-Guided Medicine, Emory University School of Medicine, Atlanta, Ga (J.K.); Division of Cardiology, Division of Electrophysiology, Emory Heart & Vascular Center at Saint Joseph's Hospital, Atlanta, Ga (A.M.P., C.M.T.); Department of Heart Failure/Transplant Cardiology, Piedmont Heart Institute, Piedmont Healthcare, Atlanta, Ga (D.W.M.); and Franklin College of Arts and Sciences, University of Georgia, Athens, Ga (F.J.P.).
Article Synopsis
- Ventricular arrhythmias (VAs) like ventricular tachycardia and ventricular fibrillation pose serious treatment challenges with high risks of morbidity and mortality, particularly in patients unresponsive to standard therapies.
- This study examined the safety and effectiveness of CT-guided left stellate ganglion cryoneurolysis (SGC) in these refractory VA patients across two hospitals, involving 17 patients with a notable reduction in defibrillation events after the procedure.
- Results showed that 82% of patients were free from defibrillation 24 hours post-procedure, with no serious adverse effects reported, highlighting the potential of SGC as a beneficial alternative treatment strategy for refractory VAs.
Spontaneous pre-excited supraventricular tachycardias in a Labrador Retriever.
J Vet Cardiol
August 2024
Clinica Veterinaria Malpensa, Viale Marconi, 27, 21017, Samarate, VA, Italy; College of Veterinary Medicine, Cornell University, 930 Campus Road, 14853, Ithaca, NY, USA.
A four-year-old Labrador Retriever was presented for intermittent tachycardia. The electrocardiogram showed sinus rhythm conducted with ventricular pre-excitation and short runs of orthodromic atrioventricular reciprocating tachycardia. Four months later, the rhythm degenerated into a symptomatic sustained tachycardia, suspected to be pre-excited atrial fibrillation, a potentially life-threatening rhythm in the presence of an accessory pathway with a short refractory period.
View Article and Find Full Text PDFExternal electrical and pharmacological cardioversion for atrial fibrillation, atrial flutter or atrial tachycardias: a network meta-analysis.
Cochrane Database Syst Rev
June 2024
Barts Heart Centre, St Bartholomew's Hospital, Barts Health NHS Trust, London, UK.
Article Synopsis
- Atrial fibrillation (AF) is a common heart rhythm disorder, and cardioversion is a method used to return the heart to its normal rhythm through medications or electrical shocks.
- The study aimed to evaluate the effectiveness and safety of both pharmacological and electrical cardioversion treatments for AF and related conditions.
- Researchers analyzed data from 112 randomized controlled trials involving nearly 16,000 patients, noting that many trials had a high risk of bias and varying demographics among participants.
Seizure suppression and neuroprotection in soman-exposed rats following delayed intramuscular treatment of adenosine A receptor agonist as an adjunct to standard medical treatment.
Toxicol Appl Pharmacol
July 2024
Neuroscience Department, Medical Toxicology Research Division, U.S. Army Medical Research Institute of Chemical Defense, Aberdeen Proving Ground, MD 21010-5400, USA. Electronic address:
Article Synopsis
- - Soman causes harmful excitotoxic effects by blocking acetylcholinesterase, leading to prolonged seizures (SSE) and brain damage in models.
- - A study tested the delayed use of the adenosine receptor agonist ENBA alongside current treatments (atropine, 2-PAM, midazolam) in rats, finding that ENBA improved seizure termination and reduced brain damage compared to midazolam alone.
- - Despite the benefits in seizure control and neuroprotection, the combination of ENBA with midazolam resulted in lower survival rates after 14 days, suggesting that ENBA may enhance midazolam's harmful effects on neurons, requiring careful dosage adjustments.
Learning and memory function preserved by delayed A adenosine receptor agonist treatment following soman intoxication in rats and a humanized esterase mouse model.
Neuropharmacology
August 2024
Medical Toxicology Research Division, U.S. Army Medical Research Institute of Chemical Defense, 8350 Ricketts Point Road, Aberdeen Proving Ground, MD, 21010-5400, USA. Electronic address:
Exposure to organophosphorus compounds, such as soman (GD), cause widespread toxic effects, sustained status epilepticus, neuropathology, and death. The A adenosine receptor agonist N-bicyclo-(2.2.
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