Accumulation of lipid peroxidation (LPO) products was investigated in external and internal membranes of mitochondria with anoxia. The increase in LPO intensity in mitochondria membranes during hypoxia was shown to be more expressed in external membranes, with an active involvement of phospholipase A2 in the process revealed. Greater LPO intensity and lability of lysosomal membranes caused by contacts with mitochondria with anoxia have been established.
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