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[Angioedema and anaphylaxis, a family story].
Rev Infirm
September 2022
Structure des urgences, Hôpital d'instruction des armées Laveran, 13384 Marseille, France.
Non-allergic angioedema has a worrying morbidity. Clinical examination is central, as C1-esterase inhibitor deficiency will not be documented in the acute phase. In the case of anaphylaxis that does not respond to adrenaline, an early diagnosis can optimise referral of the patient to a reference healthcare establishment for a specific therapeutic protocol (icatibant, C1 inhibitor) recently updated by recommendations.
View Article and Find Full Text PDFAge Characteristics and Concomitant Diseases in Patients with Angioedema.
Open Access Maced J Med Sci
February 2019
Department of Microbiology and Immunology, Faculty of Pharmacy, Medical University-Plovdiv, Plovdiv, Bulgaria.
Background: Angioneurotic oedema (AE) is an unpredictable and dangerous disease directly threatening the patient's life due to a sudden onset of upper respiratory tract obstruction. The disease is associated with various causes and triggering factors, but little is known about the conditions that accompany AE.
Aim: The study aims to determine the age-specificities and the spectrum of concomitant diseases in patients with AE.
Lethal manifestations of angioedema.
Forensic Sci Med Pathol
September 2019
Forensic Science SA, 21 Divett Place, Adelaide, 5000, Australia.
An 86-year-old woman with a history of angioedema was found dead at her home address. She had recently complained of a swollen tongue. At autopsy the tongue was grossly edematous, protruding from the mouth.
View Article and Find Full Text PDFPharmacotherapy for Angiotensin-Converting Enzyme Inhibitor-Induced Angioedema: A Systematic Review.
Otolaryngol Head Neck Surg
February 2018
1 Department of Otolaryngology-Head and Neck Surgery, School of Medicine, Tulane University, New Orleans, Louisiana, USA.
Objective Angioedema is a potentially life-threatening complication of angiotensin-converting enzyme inhibitor (ACEI) use, occurring in up to 0.5% of users. Although the pathophysiology of ACEI-induced angioedema is attributable to elevated serum bradykinin, standard management typically includes corticosteroids and antihistamines.
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