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Pancreatic fat density and post-ERCP pancreatitis: A complex relationship or a novel predictor?
Dig Liver Dis
March 2025
Gastroenterology Unit, Nazareth Hospital EMMS, Azrieli Faculty of Medicine, Bar Ilan University, Ramat Gan, Israel. Electronic address:
Intrapancreatic adipocytes and beta cell dedifferentiation in human type 2 diabetes.
Diabetologia
March 2025
Department of Endocrinology and Metabolism, Zhongshan Hospital, Fudan University, Shanghai, China.
Aims/hypothesis: Fat deposition in the pancreas is implicated in beta cell dysfunction and the progress of type 2 diabetes. However, there is limited evidence to confirm the correlation and explore how pancreatic fat links with beta cell dysfunction in human type 2 diabetes. This study aimed to examine the spatial relationship between pancreatic fat and islets in human pancreases.
View Article and Find Full Text PDFLack of maternal exposure to somatostatin leads to diet-induced insulin and leptin resistance in mouse male offspring.
J Mol Endocrinol
March 2025
L Huang, Department of Nutrition, University of California at Davis, Davis, United States.
Somatostatin (Sst) is an inhibitory regulator of many hormones. The prenatal environment impacts offspring's risk to type 2 diabetes in adulthood. However, the effect of maternal Sst deficiency on glucose and insulin metabolism in offspring and metabolic disease risk in their adult life have been poorly understood.
View Article and Find Full Text PDFSphenocentrum jollyanum (Pierre) aqueous leaf extract demonstrates anti-inflammatory and mitochondrial-restorative influences while modulating apoptosis in streptozotocin-induced diabetic rats.
J Ethnopharmacol
March 2025
Department of Biochemistry, Faculty of Science, Ekiti State University, Ekiti State, Nigeria. Electronic address:
Ethnopharmacological Relevance: Sphenocentrum jollyanum (Pierre) is a medicinal plant native to West African countries, especially Nigeria and Ghana. The leaf of S. jollyanum is a traditional therapy for diabetes, erectile dysfunction, gastrointestinal disorders, and malaria.
View Article and Find Full Text PDFOxidative stress-induced circSOD2 inhibits osteogenesis through sponging miR-29b in metabolic-associated fatty liver disease.
World J Gastroenterol
March 2025
Department of Hepatobiliary and Pancreatic Surgery, The Second Affiliated Hospital of Zhejiang University School of Medicine, Hangzhou 310009, Zhejiang Province, China.
Background: Metabolic-associated fatty liver disease (MAFLD) is characterized by lipid accumulation in hepatocytes and is closely associated with oxidative stress. Increasing clinical evidence indicates that MAFLD is linked to bone metabolic disorders, including osteoporosis. Recent studies indicate that the expression profiles of liver circular RNAs (circRNAs) are altered in MAFLD.
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